Literature DB >> 24607325

Could Na,K-ATPase play a role in potassium leakage from irradiated erythrocytes?

Grazielle Aparecida S Maia1, Vanessa Faria Cortes1, Rosy Iara Maciel de A Ribeiro2, Julio Alberto Mignaco3, Hérica de Lima Santos1, Carlos Frederico Leite Fontes4, Leandro A Barbosa5.   

Abstract

BACKGROUND: In developing countries, the access to red blood cell (RBC) irradiators is restricted. Thus, it is a common practice in blood banks to stock irradiated RBC units until they expire. The aim of this work is to elucidate the involvement of Na,K-ATPase in potassium leakage from prophylactically irradiated RBCs.
METHODS: Whole blood was collected from healthy donors, and blood concentrates were irradiated with 25Gy of γ-radiation within 24h of collection. At days 3, 5, 7, 9, 11, 14 and 28 post-irradiation, fractions were removed and centrifuged and Na,K-ATPase activity from ghost membranes was determined.
RESULTS: The inhibition of Na,K-ATPase activity in RBCs reached 12.6% by day 7 of storage and up to 50% by day 14 of storage. The addition of vitamin C prevented the irradiation-induced loss of Na,K-ATPase activity. The irradiation of RBCs provoked an increase in potassium plasma levels and a decrease in sodium plasma levels. The incubation of RBCs with ouabain did not change the sodium or potassium levels in the plasma, and the addition of vitamin C only partially prevented a decrease in sodium levels caused by irradiation.
CONCLUSION: Because the inhibition of Na,K-ATPase by ouabain did not cause potassium accumulation in the plasma, we conclude that the irradiation-induced inhibition of the pump is not a key factor driving this effect.
Copyright © 2014 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Gamma irradiation; Na,K-ATPase; RBC storage

Mesh:

Substances:

Year:  2014        PMID: 24607325     DOI: 10.1016/j.cca.2014.02.025

Source DB:  PubMed          Journal:  Clin Chim Acta        ISSN: 0009-8981            Impact factor:   3.786


  4 in total

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  4 in total

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