Literature DB >> 24588971

Membrane attack complex generation increases as a function of time in stored blood.

X Hu1, R P Patel, J A Weinberg, M B Marques, T N Ramos, S R Barnum.   

Abstract

OBJECTIVE: To determine if the complement system, a potent mediator of inflammation, contributes to haemolysis during red blood cell (RBC) storage.
BACKGROUND: RBCs in storage undergo structural and biochemical changes that may result in adverse patient outcomes post-transfusion. Complement activation on leukodepletion and during storage may contribute to the RBC storage lesion. METHODS/MATERIALS: We performed a cross-sectional analysis of aliquots of leukoreduced RBC units, stored for 1-6 weeks, for the levels of C3a, C5a, Bb, iC3b, C4d and C5b-9 [membrane attack complex (MAC)] by enzyme-linked immunosorbent assay (ELISA).
RESULTS: We observed that only MAC levels significantly increased in RBC units as a function of storage time. We also observed that the level of C5b-9 bound to RBCs increased as a function of storage time.
CONCLUSION: MAC levels increased over time, suggesting that MAC is the primary complement-mediated contributor to changes in stored RBCs. Inhibition of the terminal complement pathway may stabilise RBC functionality and extend shelf life.
© 2014 The Authors. Transfusion Medicine © 2014 British Blood Transfusion Society.

Entities:  

Keywords:  complement; extrinsic protease pathway; membrane attack complex

Mesh:

Substances:

Year:  2014        PMID: 24588971      PMCID: PMC5847664          DOI: 10.1111/tme.12109

Source DB:  PubMed          Journal:  Transfus Med        ISSN: 0958-7578            Impact factor:   2.019


  11 in total

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5.  Universal leucodepletion: an overview of some unresolved issues and the highlights of lessons learned.

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10.  Lysis of erythrocytes by complement in the absence of antibody.

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4.  Changes in Complement Levels and Activity of Red Blood Cells, Fresh Frozen Plasma, and Platelet Concentrates During Storage.

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