Literature DB >> 24560192

How T cells lose their touch.

Michael L Dustin1.   

Abstract

T cells are among the most sensitive of cells, but in this issue of Immunity, Honda et al. (2014) demonstrate that effector T cells must lose their touch within hours to protect the host from immunopathology.
Copyright © 2014 Elsevier Inc. All rights reserved.

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Year:  2014        PMID： 24560192      PMCID： PMC5615101          DOI： 10.1016/j.immuni.2014.02.001

Source DB:  PubMed          Journal:  Immunity        ISSN： 1074-7613            Impact factor:   31.745


  11 in total

1.  T-cell priming by dendritic cells in lymph nodes occurs in three distinct phases.

Authors:  Thorsten R Mempel; Sarah E Henrickson; Ulrich H Von Andrian
Journal:  Nature       Date:  2004-01-08       Impact factor: 49.962

2.  Strength of PD-1 signaling differentially affects T-cell effector functions.

Authors:  Fang Wei; Shi Zhong; Zhengyu Ma; Hong Kong; Andrew Medvec; Rafi Ahmed; Gordon J Freeman; Michelle Krogsgaard; James L Riley
Journal:  Proc Natl Acad Sci U S A       Date:  2013-04-22       Impact factor: 11.205

3.  Tuning of antigen sensitivity by T cell receptor-dependent negative feedback controls T cell effector function in inflamed tissues.

Authors:  Tetsuya Honda; Jackson G Egen; Tim Lämmermann; Wolfgang Kastenmüller; Parizad Torabi-Parizi; Ronald N Germain
Journal:  Immunity       Date:  2014-01-16       Impact factor: 31.745

4.  T cells use two directionally distinct pathways for cytokine secretion.

Authors:  Morgan Huse; Björn F Lillemeier; Michael S Kuhns; Daniel S Chen; Mark M Davis
Journal:  Nat Immunol       Date:  2006-01-29       Impact factor: 25.606

5.  Imaging of effector memory T cells during a delayed-type hypersensitivity reaction and suppression by Kv1.3 channel block.

Authors:  Melanie P Matheu; Christine Beeton; Adriana Garcia; Victor Chi; Srikant Rangaraju; Olga Safrina; Kevin Monaghan; Marc I Uemura; Dan Li; Sukumar Pal; Luis M de la Maza; Edwin Monuki; Alexander Flügel; Michael W Pennington; Ian Parker; K George Chandy; Michael D Cahalan
Journal:  Immunity       Date:  2008-10-02       Impact factor: 31.745

6.  Programmed death-1 ligand 1 interacts specifically with the B7-1 costimulatory molecule to inhibit T cell responses.

Authors:  Manish J Butte; Mary E Keir; Theresa B Phamduy; Arlene H Sharpe; Gordon J Freeman
Journal:  Immunity       Date:  2007-07-12       Impact factor: 31.745

7.  Suppressing T cell motility induced by anti-CTLA-4 monotherapy improves antitumor effects.

Authors:  Maria Grazia Ruocco; Karsten A Pilones; Noriko Kawashima; Michael Cammer; Julie Huang; James S Babb; Mengling Liu; Silvia C Formenti; Michael L Dustin; Sandra Demaria
Journal:  J Clin Invest       Date:  2012-09-04       Impact factor: 14.808

8.  Programmed cell death 1 forms negative costimulatory microclusters that directly inhibit T cell receptor signaling by recruiting phosphatase SHP2.

Authors:  Tadashi Yokosuka; Masako Takamatsu; Wakana Kobayashi-Imanishi; Akiko Hashimoto-Tane; Miyuki Azuma; Takashi Saito
Journal:  J Exp Med       Date:  2012-05-28       Impact factor: 14.307

9.  Interactions between PD-1 and PD-L1 promote tolerance by blocking the TCR-induced stop signal.

Authors:  Brian T Fife; Kristen E Pauken; Todd N Eagar; Takashi Obu; Jenny Wu; Qizhi Tang; Miyuki Azuma; Matthew F Krummel; Jeffrey A Bluestone
Journal:  Nat Immunol       Date:  2009-09-27       Impact factor: 25.606

10.  PD-1 promotes immune exhaustion by inducing antiviral T cell motility paralysis.

Authors:  Bernd H Zinselmeyer; Sara Heydari; Catarina Sacristán; Debasis Nayak; Michael Cammer; Jasmin Herz; Xiaoxiao Cheng; Simon J Davis; Michael L Dustin; Dorian B McGavern
Journal:  J Exp Med       Date:  2013-03-25       Impact factor: 14.307
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