Literature DB >> 24506861

High-density lipoproteins put out the fire.

Kathryn J Moore1, Edward A Fisher2.   

Abstract

Macrophages in atherosclerotic plaques are activated, inflammatory cells that directly contribute to the disease process. De Nardo et al. (2013), now report that high-density lipoproteins (HDL) can reprogram macrophages to be less inflammatory through an ATF3-dependent pathway, providing another mechanistic basis for the atheroprotective properties of HDL.
Copyright © 2014 Elsevier Inc. All rights reserved.

Entities:  

Mesh：

Substances：

Year:  2014        PMID： 24506861      PMCID： PMC3962668          DOI： 10.1016/j.cmet.2014.01.009

Source DB:  PubMed          Journal:  Cell Metab        ISSN： 1550-4131            Impact factor:   27.287


  6 in total

Review 1.  Subendothelial lipoprotein retention as the initiating process in atherosclerosis: update and therapeutic implications.

Authors:  Ira Tabas; Kevin Jon Williams; Jan Borén
Journal:  Circulation       Date:  2007-10-16       Impact factor: 29.690

2.  HDL promotes rapid atherosclerosis regression in mice and alters inflammatory properties of plaque monocyte-derived cells.

Authors:  Jonathan E Feig; James X Rong; Raanan Shamir; Marie Sanson; Yuliya Vengrenyuk; Jianhua Liu; Katey Rayner; Kathryn Moore; Michael Garabedian; Edward A Fisher
Journal:  Proc Natl Acad Sci U S A       Date:  2011-04-11       Impact factor: 11.205

3.  Antagonism of miR-33 in mice promotes reverse cholesterol transport and regression of atherosclerosis.

Authors:  Katey J Rayner; Frederick J Sheedy; Christine C Esau; Farah N Hussain; Ryan E Temel; Saj Parathath; Janine M van Gils; Alistair J Rayner; Aaron N Chang; Yajaira Suarez; Carlos Fernandez-Hernando; Edward A Fisher; Kathryn J Moore
Journal:  J Clin Invest       Date:  2011-06-06       Impact factor: 14.808

4.  Increased inflammatory gene expression in ABC transporter-deficient macrophages: free cholesterol accumulation, increased signaling via toll-like receptors, and neutrophil infiltration of atherosclerotic lesions.

Authors:  Laurent Yvan-Charvet; Carrie Welch; Tamara A Pagler; Mollie Ranalletta; Mohamed Lamkanfi; Seongah Han; Minako Ishibashi; Rong Li; Nan Wang; Alan R Tall
Journal:  Circulation       Date:  2008-10-13       Impact factor: 29.690

5.  HDL induces the expression of the M2 macrophage markers arginase 1 and Fizz-1 in a STAT6-dependent process.

Authors:  Marie Sanson; Emilie Distel; Edward A Fisher
Journal:  PLoS One       Date:  2013-08-21       Impact factor: 3.240

6.  High-density lipoprotein mediates anti-inflammatory reprogramming of macrophages via the transcriptional regulator ATF3.

Authors:  Dominic De Nardo; Larisa I Labzin; Hajime Kono; Reiko Seki; Susanne V Schmidt; Marc Beyer; Dakang Xu; Sebastian Zimmer; Catharina Lahrmann; Frank A Schildberg; Johanna Vogelhuber; Michael Kraut; Thomas Ulas; Anja Kerksiek; Wolfgang Krebs; Niklas Bode; Alena Grebe; Michael L Fitzgerald; Nicholas J Hernandez; Bryan R G Williams; Percy Knolle; Manfred Kneilling; Martin Röcken; Dieter Lütjohann; Samuel D Wright; Joachim L Schultze; Eicke Latz
Journal:  Nat Immunol       Date:  2013-12-08       Impact factor: 25.606
  6 in total
  2 in total

1.  Apoptotic cells trigger a membrane-initiated pathway to increase ABCA1.

Authors:  Aaron M Fond; Chang Sup Lee; Ira G Schulman; Robert S Kiss; Kodi S Ravichandran
Journal:  J Clin Invest       Date:  2015-06-15       Impact factor: 14.808

Review 2.  Activating transcription factor 3, an early cellular adaptive responder in ischemia/reperfusion-induced injury.

Authors:  Heng Lin; Ching-Feng Cheng
Journal:  Ci Ji Yi Xue Za Zhi       Date:  2018 Apr-Jun
  2 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.