Literature DB >> 24506563

The decrease of NAD(P)H:quinone oxidoreductase 1 activity and increase of ROS production by NADPH oxidases are early biomarkers in doxorubicin cardiotoxicity.

Ricardo Lagoa1, Carlos Gañán, Carmen López-Sánchez, Virginio García-Martínez, Carlos Gutierrez-Merino.   

Abstract

CONTEXT: Doxorubicin cardiotoxicity displays a complex and multifactorial progression.
OBJECTIVE: Identify early biochemical mechanisms leading to a sustained imbalance of cellular bioenergetics.
METHODS: Measurements of the temporal evolution of selected biochemical markers after treatment of rats with doxorubicin (20 mg/kg body weight).
RESULTS: Doxorubicin treatment increased lipid oxidation, catalase activity and production of H₂O₂ by Nox-NADPH oxidases, and down-regulated NAD(P)H: quinone oxidoreductase-1 prior eliciting changes in reduced glutathione, protein carbonyls and protein nitrotyrosines. Alterations of mitochondrial and myofibrillar bioenergetics biomarkers were detected only after this oxidative imbalance was established. NAD(P)H: quinone oxidoreductase-1 activity and increase of hydrogen peroxide production by NADPH oxidases are early biomarkers in doxorubicin cardiotoxicity.

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Year:  2014        PMID: 24506563     DOI: 10.3109/1354750X.2014.885084

Source DB:  PubMed          Journal:  Biomarkers        ISSN: 1354-750X            Impact factor:   2.658


  6 in total

1.  The interplay between genetic background and sexual dimorphism of doxorubicin-induced cardiotoxicity.

Authors:  Beshay N Zordoky; M Judith Radin; Lois Heller; Anthony Tobias; Ilze Matise; Fred S Apple; Sylvia A McCune; Leslie C Sharkey
Journal:  Cardiooncology       Date:  2016-03-15

2.  Possible roles of genetic variations in chemotherapy related cardiotoxicity in pediatric acute lymphoblastic leukemia and osteosarcoma.

Authors:  Judit C Sági; Bálint Egyed; Andrea Kelemen; Nóra Kutszegi; Márta Hegyi; András Gézsi; Martina Ayaka Herlitschke; Andrea Rzepiel; Lili E Fodor; Gábor Ottóffy; Gábor T Kovács; Dániel J Erdélyi; Csaba Szalai; Ágnes F Semsei
Journal:  BMC Cancer       Date:  2018-07-03       Impact factor: 4.430

3.  Punicalagin protects H9c2 cardiomyocytes from doxorubicin-induced toxicity through activation of Nrf2/HO-1 signaling.

Authors:  Mingfang Ye; Linlin Zhang; Yuanming Yan; Huizhong Lin
Journal:  Biosci Rep       Date:  2019-05-14       Impact factor: 3.840

4.  Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment.

Authors:  Fabio Ntagwabira; Madison Trujillo; Taylor McElroy; Taurean Brown; Pilar Simmons; Delawerence Sykes; Antiño R Allen
Journal:  Int J Mol Sci       Date:  2022-02-11       Impact factor: 5.923

Review 5.  Role of Drug Metabolism in the Cytotoxicity and Clinical Efficacy of Anthracyclines.

Authors:  Derek W Edwardson; Rashmi Narendrula; Simon Chewchuk; Kyle Mispel-Beyer; Jonathan P J Mapletoft; Amadeo M Parissenti
Journal:  Curr Drug Metab       Date:  2015       Impact factor: 3.731

6.  Klotho Improves Cardiac Function by Suppressing Reactive Oxygen Species (ROS) Mediated Apoptosis by Modulating Mapks/Nrf2 Signaling in Doxorubicin-Induced Cardiotoxicity.

Authors:  Huolan Zhu; Yan Gao; Shunming Zhu; Qianwei Cui; Jie Du
Journal:  Med Sci Monit       Date:  2017-11-06
  6 in total

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