Joon-Yong Jung1, Won-Hee Jee1, Sung Hwan Hong2, Heung Sik Kang3, Hye Won Chung4, Kyung-Nam Ryu5, Jee-Young Kim1, Soo-A Im1, Jeong-Mi Park1, Mi-Sook Sung1, Yeon-Soo Lee1, Suk-Joo Hong6, Chan-Kwon Jung7, Yang-Guk Chung8. 1. Department of Radiology, College of Medicine, The Catholic University of Korea, Seoul 137-701, Korea. 2. Department of Radiology, Seoul National University College of Medicine, Seoul 110-744, Korea. 3. Department of Radiology, Seoul National University Bundang Hospital, Seongnam 463-707, Korea. 4. Department of Radiology, Asan Medical Center, University of Ulsan College of Medicine, Seoul 138-736, Korea. 5. Department of Radiology, Kyung Hee University College of Medicine, Seoul 130-872, Korea. 6. Department of Radiology, Korea University College of Medicine, Seoul 136-705, Korea. 7. Department of Pathology, College of Medicine, The Catholic University of Korea, Seoul 137-701, Korea. 8. Department of Orthopedic Surgery, College of Medicine, The Catholic University of Korea, Seoul 137-701, Korea.
Abstract
OBJECTIVE: The aim of this study was to describe MR findings of osteofibrous dysplasia. MATERIALS AND METHODS: MR images of 24 pathologically proven osteofibrous dysplasia cases were retrospectively analyzed for a signal intensity of the lesion, presence of intralesional fat signal, internal hypointense band, multilocular appearance, cortical expansion, intramedullary extension, cystic area, cortical breakage and extraosseous extension, abnormal signal from the adjacent bone marrow and soft tissue and patterns of contrast enhancement. RESULTS: All cases of osteofibrous dysplasia exhibited intermediate signal intensity on T1-weighted images. On T2-weighted images, 20 and 4 cases exhibited heterogeneously intermediate and high signal intensity, respectively. Intralesional fat was identified in 12% of the cases. Internal low-signal bands and multilocular appearance were observed in 91%. Cortical expansion was present in 58%. Intramedullary extension was present in all cases, and an entire intramedullary replacement was observed in 33%. Cortical breakage (n = 3) and extraosseous mass formation (n = 1) were observed in cases with pathologic fractures only. A cystic area was observed in one case. Among 21 cases without a pathologic fracture, abnormal signal intensity in the surrounding bone marrow and adjacent soft tissue was observed in 43% and 48%, respectively. All cases exhibited diffuse contrast enhancement. CONCLUSION: Osteofibrous dysplasia exhibited diverse imaging features ranging from lesions confined to the cortex to more aggressive lesions with complete intramedullary involvement or perilesional marrow edema.
OBJECTIVE: The aim of this study was to describe MR findings of osteofibrous dysplasia. MATERIALS AND METHODS: MR images of 24 pathologically proven osteofibrous dysplasia cases were retrospectively analyzed for a signal intensity of the lesion, presence of intralesional fat signal, internal hypointense band, multilocular appearance, cortical expansion, intramedullary extension, cystic area, cortical breakage and extraosseous extension, abnormal signal from the adjacent bone marrow and soft tissue and patterns of contrast enhancement. RESULTS: All cases of osteofibrous dysplasia exhibited intermediate signal intensity on T1-weighted images. On T2-weighted images, 20 and 4 cases exhibited heterogeneously intermediate and high signal intensity, respectively. Intralesional fat was identified in 12% of the cases. Internal low-signal bands and multilocular appearance were observed in 91%. Cortical expansion was present in 58%. Intramedullary extension was present in all cases, and an entire intramedullary replacement was observed in 33%. Cortical breakage (n = 3) and extraosseous mass formation (n = 1) were observed in cases with pathologic fractures only. A cystic area was observed in one case. Among 21 cases without a pathologic fracture, abnormal signal intensity in the surrounding bone marrow and adjacent soft tissue was observed in 43% and 48%, respectively. All cases exhibited diffuse contrast enhancement. CONCLUSION:Osteofibrous dysplasia exhibited diverse imaging features ranging from lesions confined to the cortex to more aggressive lesions with complete intramedullary involvement or perilesional marrow edema.
Entities:
Keywords:
Bone neoplasms; Magnetic resonance imaging; Osteofibrous dysplasia
Authors: W H Jee; B Y Choe; H S Kang; K J Suh; J S Suh; K N Ryu; Y S Lee; I Y Ok; J M Kim; K H Choi; K S Shinn Journal: Radiology Date: 1998-10 Impact factor: 11.105
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