Literature DB >> 24463479

Usefulness of frataxin immunoassays for the diagnosis of Friedreich ataxia.

Eric C Deutsch1, Devin Oglesbee2, Nathaniel R Greeley3, David R Lynch1.   

Abstract

BACKGROUND: Friedreich ataxia (FRDA) is a neurodegenerative disease caused by mutations in the frataxin (FXN) gene, resulting in reduced expression of the mitochondrial protein frataxin. Improved understanding of the pathophysiology of the disease has led to a growing need for informative biomarkers to assess disease progression and response to therapeutic intervention.
OBJECTIVE: To evaluate the performance of frataxin measurements as a diagnostic tool using two different immunoassays.
METHODS: Clinical and demographic information was provided through an ongoing longitudinal natural history study on FRDA. Frataxin protein levels from multiple cell types in controls, carriers and FRDA patients were measured and compared using a lateral flow immunoassay and a Luminex xMAP-based immunoassay. Receiver operating characteristic curve analyses were then performed to evaluate the sensitivity, specificity, and positive and negative predictive values for each immunoassay.
RESULTS: For whole blood and buccal cells, analysing FRDA patients and carriers together in a cohort resulted in higher sensitivities and positive predictive values compared with analyzing controls and carriers together, with similar results between each tissue type. We then compared the usefulness of a lateral flow immunoassay with a multianalyte Luminex xMAP-based immunoassay, and showed that both assays demonstrate high positive predictive values with low rates of false negatives and false positives.
CONCLUSIONS: Frataxin measurements from peripheral tissues can be used to identify FRDA patients and carriers. While multiple cell types and assays may be useful for diagnostic purposes, each assay and cell type used has its advantages and disadvantages depending on study design and scope. Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions.

Entities:  

Keywords:  MITOCHONDRIAL DISORDERS; MOVEMENT DISORDERS; PAEDIATRIC

Mesh:

Substances:

Year:  2014        PMID: 24463479     DOI: 10.1136/jnnp-2013-306788

Source DB:  PubMed          Journal:  J Neurol Neurosurg Psychiatry        ISSN: 0022-3050            Impact factor:   10.154


  8 in total

1.  Stable isotopes and LC-MS for monitoring metabolic disturbances in Friedreich's ataxia platelets.

Authors:  Andrew J Worth; Sankha S Basu; Eric C Deutsch; Wei-Ting Hwang; Nathaniel W Snyder; David R Lynch; Ian A Blair
Journal:  Bioanalysis       Date:  2015       Impact factor: 2.681

Review 2.  Three Adult-Onset Autosomal Recessive Ataxias: What Adult Neurologists Need to Know.

Authors:  Jordan A Paulus-Andres; Melinda S Burnett
Journal:  Neurol Clin Pract       Date:  2021-06

3.  Simultaneous Quantification of Mitochondrial Mature Frataxin and Extra-Mitochondrial Frataxin Isoform E in Friedreich's Ataxia Blood.

Authors:  Qingqing Wang; Laurent Laboureur; Liwei Weng; Nicolas M Eskenazi; Lauren A Hauser; Clementina Mesaros; David R Lynch; Ian A Blair
Journal:  Front Neurosci       Date:  2022-04-28       Impact factor: 5.152

4.  Clinical Evidence for Variegated Silencing in Patients With Friedreich Ataxia.

Authors:  Layne N Rodden; Christian Rummey; Yi Na Dong; David R Lynch
Journal:  Neurol Genet       Date:  2022-05-17

5.  Friedreich ataxia in Norway - an epidemiological, molecular and clinical study.

Authors:  Iselin Marie Wedding; Mette Kroken; Sandra Pilar Henriksen; Kaja Kristine Selmer; Torunn Fiskerstrand; Per Morten Knappskog; Tone Berge; Chantal M E Tallaksen
Journal:  Orphanet J Rare Dis       Date:  2015-09-04       Impact factor: 4.123

Review 6.  Role of frataxin protein deficiency and metabolic dysfunction in Friedreich ataxia, an autosomal recessive mitochondrial disease.

Authors:  Elisia Clark; Joseph Johnson; Yi Na Dong; Elizabeth Mercado-Ayon; Nathan Warren; Mattieu Zhai; Emily McMillan; Amy Salovin; Hong Lin; David R Lynch
Journal:  Neuronal Signal       Date:  2018-11-02

7.  Randomized, double-blind, placebo-controlled study of interferon-γ 1b in Friedreich Ataxia.

Authors:  David R Lynch; Lauren Hauser; Ashley McCormick; McKenzie Wells; Yi Na Dong; Shana McCormack; Kim Schadt; Susan Perlman; Sub H Subramony; Katherine D Mathews; Alicia Brocht; Julie Ball; Renee Perdok; Amy Grahn; Tom Vescio; Jeffrey W Sherman; Jennifer M Farmer
Journal:  Ann Clin Transl Neurol       Date:  2019-02-27       Impact factor: 4.511

8.  Perturbation of cellular proteostasis networks identifies pathways that modulate precursor and intermediate but not mature levels of frataxin.

Authors:  Joseph F Nabhan; Renea L Gooch; Eugene L Piatnitski Chekler; Betsy Pierce; Christine E Bulawa
Journal:  Sci Rep       Date:  2015-12-16       Impact factor: 4.379

  8 in total

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