Literature DB >> 24398014

Cystic fibrosis transmembrane regulator (CFTR) in human trophoblast BeWo cells and its relation to cell migration.

G I Marino1, B A Kotsias2.   

Abstract

INTRODUCTION: ENaC and CFTR are coexpressed in epithelia and have positive or negative functional interactions. In addition, ENaC and CFTR promote migration in placental trophoblastic cells and human airway cells, respectively. Here we tested the idea if CFTR is functionally expressed in BeWo cells, a trophoblastic cell line, and if it is involved in their migratory behavior.
METHODS: CFTR expression was studied in BeWo cells with RT-PCR, biotinylation and Western blot. Ion currents were analyzed with patch clamp, and cell migration with the wound healing method.
RESULTS: The mature CFTR 160-kDa band was present, and its localization at the surface membrane was confirmed. Forskolin (20 μM), an adenylate cyclase activator, was used for channel activation, and subsequently CFTR(inh)-172 (2 μM) for its inhibition. The conductances in the presence of CFTR(inh)-172 plus forskolin (16.0 ± 0.7 pS/pF and 32.6 ± 1.5 pS/pF) were significantly lower than in presence of only forskolin (29.7 ± 0.9 and 47.0 ± 2.0 pS/pF). The conductance of CFTR(inh)-172 inhibited currents was 14.9 ± 0.7 pS/pF with a linear I-V relationship illustrating the nonrectifying properties of the CFTR. Cell migration was measured and covered 11.2 ± 0.4, 24.0 ± 1.7 and 13.9 ± 1.0% of the wound when cells were cultivated under control, forskolin, and forskolin plus CFTR(inh)-172, respectively. Proliferation was not changed by any of the treatments.
CONCLUSIONS: Our results shows that BeWo cells functionally express the CFTR which plays a role in the wound healing increasing the cell migration process.
Copyright © 2013 Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  BeWo cells; CFTR; CFTR(inh)-172; Forskolin; Ion currents; Wound healing

Mesh:

Substances:

Year:  2013        PMID: 24398014     DOI: 10.1016/j.placenta.2013.12.004

Source DB:  PubMed          Journal:  Placenta        ISSN: 0143-4004            Impact factor:   3.481


  7 in total

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Journal:  Crit Care Med       Date:  2014-11       Impact factor: 7.598

2.  Hyperoxia treatment of TREK-1/TREK-2/TRAAK-deficient mice is associated with a reduction in surfactant proteins.

Authors:  Andreas Schwingshackl; Benjamin Lopez; Bin Teng; Charlean Luellen; Florian Lesage; John Belperio; Riccardo Olcese; Christopher M Waters
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3.  CFTR channel in oocytes from Xenopus laevis and its regulation by xShroom1 protein.

Authors:  Alejandra G Palma; Luciano Galizia; Basilio A Kotsias; Gabriela I Marino
Journal:  Pflugers Arch       Date:  2016-02-18       Impact factor: 3.657

Review 4.  What Role Does CFTR Play in Development, Differentiation, Regeneration and Cancer?

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Journal:  Int J Mol Sci       Date:  2020-04-29       Impact factor: 5.923

5.  Functional relationship between CFTR and RAC3 expression for maintaining cancer cell stemness in human colorectal cancer.

Authors:  Alejandra Graciela Palma; Mileni Soares Machado; María Cecilia Lira; Francisco Rosa; María Fernanda Rubio; Gabriela Marino; Basilio Aristidis Kotsias; Mónica Alejandra Costas
Journal:  Cell Oncol (Dordr)       Date:  2021-02-22       Impact factor: 6.730

Review 6.  ENaC/DEG in Tumor Development and Progression.

Authors:  Cui Liu; Li-Li Zhu; Si-Guang Xu; Hong-Long Ji; Xiu-Min Li
Journal:  J Cancer       Date:  2016-09-13       Impact factor: 4.207

7.  Upregulation of CFTR in patients with endometriosis and its involvement in NFκB-uPAR dependent cell migration.

Authors:  Wenqing Huang; Aihong Jin; Jieting Zhang; Chaoqun Wang; Lai Ling Tsang; Zhiming Cai; Xiaping Zhou; Hao Chen; Hsiao Chang Chan
Journal:  Oncotarget       Date:  2017-03-22
  7 in total

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