Literature DB >> 24324081

Antibody against tumor necrosis factor-α reduces bortezomib-induced allodynia in a rat model.

Alessia Chiorazzi1, Annalisa Canta, Cristina Meregalli, Valentina Carozzi, Barbara Sala, Norberto Oggioni, Johan Monbaliu, Helgi VAN DE Velde, Guido Cavaletti.   

Abstract

BACKGROUND: Bortezomib is an anti-neoplastic drug acting against multiple myeloma but its use is associated with the onset of painful peripheral neuropathy. Tumor necrosis factor-α (TNFα) is associated with the development of neuropathic pain; several models have shown that the inactivation of TNFα leads to a reduction in pain stimuli perception. The aim of the present study was to analyze if the administration of an antibody against TNFα is able to prevent the development of bortezomib-induced neuropathic pain.
MATERIALS AND METHODS: Nerve conduction velocity was measured and a histopathological examination was performed to assess the extent of peripheral neuropathy. To study the onset of painful neuropathy, the response to mechanical or thermal stimuli was evaluated.
RESULTS: This study demonstrated that co-administration of an antibody against TNFα is able to prevent allodynia induced by bortezomib but does not reduce neuropathy.
CONCLUSION: Targeting TNFα might be useful in limiting patients' discomfort during bortezomib therapy.

Entities:  

Keywords:  Bortezomib-induced neuropathy; allodynia; anti-TNFα; tumor necrosis factor-α

Mesh:

Substances:

Year:  2013        PMID: 24324081

Source DB:  PubMed          Journal:  Anticancer Res        ISSN: 0250-7005            Impact factor:   2.480


  9 in total

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