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Literature DB >> 24315365

Was it something I ate?

Alastair S Garfield¹, Bradford B Lowell.

Abstract

Aversive visceral stimuli, such as those associated with sickness, suppress appetite. Yet an understanding of the neural mechanisms underlying illness-related anorexia has remained elusive. Carter et al. (2013) now identify a specific hindbrain → amygdala circuit that contributes to illness-induced loss of appetite.

Entities: Chemical Disease Gene Species

Mesh：

Year: 2013 PMID： 24315365 PMCID： PMC3887394 DOI： 10.1016/j.cmet.2013.11.010

Source DB: PubMed Journal: Cell Metab ISSN： 1550-4131 Impact factor: 27.287

9 in total

1. NPY/AgRP neurons are essential for feeding in adult mice but can be ablated in neonates.

Authors: Serge Luquet; Francisco A Perez; Thomas S Hnasko; Richard D Palmiter
Journal: Science Date: 2005-10-28 Impact factor: 47.728

2. Rapid, reversible activation of AgRP neurons drives feeding behavior in mice.

Authors: Michael J Krashes; Shuichi Koda; ChianPing Ye; Sarah C Rogan; Andrew C Adams; Daniel S Cusher; Eleftheria Maratos-Flier; Bryan L Roth; Bradford B Lowell
Journal: J Clin Invest Date: 2011-04 Impact factor: 14.808

3. Ablation of neurons expressing agouti-related protein activates fos and gliosis in postsynaptic target regions.

Authors: Qi Wu; Maureen P Howell; Richard D Palmiter
Journal: J Neurosci Date: 2008-09-10 Impact factor: 6.167

Review 4. The amygdala and persistent pain.

Authors: Volker Neugebauer; Weidong Li; Gary C Bird; Jeong S Han
Journal: Neuroscientist Date: 2004-06 Impact factor: 7.519

1 in total

1. Parabrachial CGRP Neurons Control Meal Termination.

Authors: Carlos A Campos; Anna J Bowen; Michael W Schwartz; Richard D Palmiter
Journal: Cell Metab Date: 2016-05-10 Impact factor: 27.287

1 in total

Was it something I ate?

1. NPY/AgRP neurons are essential for feeding in adult mice but can be ablated in neonates.

2. Rapid, reversible activation of AgRP neurons drives feeding behavior in mice.

3. Ablation of neurons expressing agouti-related protein activates fos and gliosis in postsynaptic target regions.

Review 4. The amygdala and persistent pain.

5. Loss of GABAergic signaling by AgRP neurons to the parabrachial nucleus leads to starvation.

6. Deconstruction of a neural circuit for hunger.

7. Arcuate AgRP neurons and the regulation of energy balance.

8. AGRP neurons are sufficient to orchestrate feeding behavior rapidly and without training.

9. Genetic identification of a neural circuit that suppresses appetite.

1. Parabrachial CGRP Neurons Control Meal Termination.