Literature DB >> 24291343

Characterization and validation of new tools for measuring site-specific cardiac troponin I phosphorylation.

Stephen F Thoemmes1, Crystal A Stutzke2, Yanmei Du1, Michael D Browning2, Peter M Buttrick1, Lori A Walker3.   

Abstract

Phosphorylation of cardiac troponin I is a well established mechanism by which cardiac contractility is modulated. However, there are a number of phosphorylation sites on TnI which contribute singly or in combination to influence cardiac function. Accordingly, methods for accurately measuring site-specific TnI phosphorylation are needed. Currently, two strategies are employed: mass spectrometry, which is costly, difficult and has a low throughput; and Western blotting using phospho-specific antibodies, which is limited by the availability of reagents. In this report, we describe a cohort of new site-specific TnI phosphoantibodies, generated against physiologically relevant phosphorylation sites, that are superior to the current commercially available antibodies: to phospho-serine 22/23 which shows a >5-fold phospho-specificity for phosphorylated TnI; to phospho-serine 43, which has >3-fold phospho-specificity for phosphorylated TnI; and phospho-serine 150 which has >2-fold phospho-specificity for phosphorylated TnI. These new antibodies demonstrated greater sensitivity and specificity for the phosphorylated TnI than the most widely used commercially available reagents. For example, at a protein load of 20 μg of total cardiac extract, a commercially available antibody recognized both phosphorylated and dephosphorylated TnI to the same degree. At the same protein load our phospho-serine 22/23 antibody exhibited no cross-reactivity with dephosphorylated TnI. These new tools should allow a more accurate assessment and a better understanding of the role of TnI phosphorylation in the response of the heart to pathologic stress.
Copyright © 2013 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  Antibodies; Cardiac; Cardiovascular disease; Human; Phosphorylation; Troponin I

Mesh:

Substances:

Year:  2013        PMID: 24291343      PMCID: PMC3945064          DOI: 10.1016/j.jim.2013.11.020

Source DB:  PubMed          Journal:  J Immunol Methods        ISSN: 0022-1759            Impact factor:   2.303


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