Literature DB >> 24282904

Expression of NFkappaB, ICAM1, and VCAM1 in rheumatic heart disease with atrial fibrillation.

Li-Ping Chen1, Hui Liu, Yuan Huang, Xiang-Yan Zhang, Riley E Alexander, Liang Cheng.   

Abstract

OBJECTIVE: To examine the expression of nuclear factor kappaB (NFkappaB), intercellular adhesion molecule 1 (ICAM1), and vascular cell adhesion molecule 1 (VCAM1) levels in rheumatic heart disease patients who suffer from atrial fibrillation and those who do not. STUDY
DESIGN: Twenty-six patients with rheumatic heart disease were enrolled. Ten patients had no history of atrial fibrillation and 16 had atrial fibrillation. Atrial tissue was obtained from the right atrial appendage during heart surgery. The expression levels of NFkappaB, ICAM1, and VCAM1 were examined with immunohistochemical staining.
RESULTS: Myocardial inflammation and fibrosis were both increased in patients with atrial fibrillation. The levels of NFkappaB, ICAM1, and VCAM1 were significantly elevated in patients with atrial fibrillation as compared with the control group (all p < 0.05).
CONCLUSION: Overexpression of NFkappaB, ICAM1, and VCAM1 may be involved in the development and maintenance of atrial fibrillation in patients with rheumatic heart disease.

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Year:  2013        PMID: 24282904

Source DB:  PubMed          Journal:  Anal Quant Cytopathol Histpathol        ISSN: 2578-742X


  1 in total

1.  Exercise Attenuates Acute β-Adrenergic Overactivation-Induced Cardiac Fibrosis by Modulating Cytokines.

Authors:  Akehu Alemasi; Ning Cao; Xiangbo An; Jimin Wu; Huijun Gu; Haiyi Yu; Yao Song; Huan Wang; Youyi Zhang; Han Xiao; Wei Gao
Journal:  J Cardiovasc Transl Res       Date:  2019-06-03       Impact factor: 4.132

  1 in total

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