Problems of interpreting results obtained in studies of somaclonal variation in gliadin proteins in wheat.

It is quite likely that somacloning increases the frequency of mutations: this has, in fact, been claimed for gliadin proteins. However, in such instances it is important to distinguish between true mutations and presumed changes in regenerants due to cross pollination or admixture. We present herein arguments that some so-claimed somaclonal variants in gliadincoding genes are unlikely to be due to mutations since they would have had to involve directed changes at several loci simultaneously.