Literature DB >> 24187610

Shell-crosslinked knedel-like nanoparticles induce lower immunotoxicity than their non-crosslinked analogs.

Mahmoud Elsabahy1, Sandani Samarajeewa, Jeffery E Raymond, Corrie Clark, Karen L Wooley.   

Abstract

The development of stable nanoparticles that can withstand the changing conditions experienced in a biological setting and also be of low n class="Disease">toxicity anpan>d immunpan>ogenicity is of particular impan> class="Gene">portance to address the problems associated with currently utilized nanotechnology-based therapeutics and diagnostics. The use of crosslinked nanoparticles continues to receive special impetus, due to their robust structure and high kinetic stability, and they have recently been shown to induce lower cytotoxicity than their non-crosslinked micellar counterparts. In the current study, poly(acrylamidoethylamine)-block-poly(DL-lactide) (PAEA90-b-PDLLA40) copolymers were synthesized, self-assembled in water to yield nanoscopic polymeric micelles, and the effects of decorating the micellar surface with poly(ethylene glycol) (i.e. PEGylation) and crosslinking the PAEA layer to varying extents on the physicochemical characteristics, cytotoxicity and immunotoxicity of the nanoparticles were studied. Herein, we report for the first time that crosslinking can efficiently reduce the immunotoxicity of polymeric nanomaterials. In addition, increasing the degree of crosslinking further reduced the accessibility of biomolecules to the core of the nanoparticles and decreased their cytotoxicity and immunotoxicity. It is also highlighted that crosslinking can be more efficient than PEGylation in reducing the immunotoxicity of nanomaterials. Shell-crosslinking of block copolymer micelles, therefore, is expected to advance their clinical development beyond the earlier known effects, and to broaden the implications in the field of nanomedicine.

Entities:  

Keywords:  Shell-crosslinked knedel-like nanoparticles; crosslinking; cytokines; immunotoxicity; poly(ethylene glycol); toxicity

Year:  2013        PMID: 24187610      PMCID: PMC3811931          DOI: 10.1039/C3TB20668H

Source DB:  PubMed          Journal:  J Mater Chem B        ISSN: 2050-750X            Impact factor:   6.331


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