Furosemide blocks the apomorphine-elicited Cl-channel activity of rat striatal dopamine receptors functionally reconstituted into bimolecular lipid membrane.

Furosemide blocks the apomorphine-induced step-like conductance increase of the bimolecular lipid membrane pretreated with rat striatal homogenate. Biionic potential measurements also suggest that the reconstituted dopamine receptor is probably linked to a chloride channel.