Literature DB >> 24114429

A study of N-methyl-D-aspartate receptor gene (GRIN2B) variants as predictors of treatment-resistant major depression.

Chen Zhang1, Zezhi Li, Zhiguo Wu, Jun Chen, Zuowei Wang, Daihui Peng, Wu Hong, Chengmei Yuan, Zhen Wang, Shunying Yu, Yifeng Xu, Lin Xu, Zeping Xiao, Yiru Fang.   

Abstract

RATIONALE: In clinical practice, ketamine, an antagonist of the N-methyl-D-aspartate receptor (NMDAR), is used to alleviate depressive symptoms in patients with major depressive disorder (MDD), especially in those with treatment-resistant depression (TRD). Accordingly, the human gene coding for the 2B subunit of the NMDAR (GRIN2B) is considered a promising candidate gene for MDD susceptibility.
OBJECTIVES: The primary aim of this study is to examine whether potentially functional polymorphisms of GRIN2B confer risk for MDD, and second to investigate whether GRIN2B acts as a genetic predictor for TRD in MDD patients.
METHODS: We enrolled 178 TRD and 612 non-TRD patients as well as 779 healthy controls.
RESULTS: Four potentially functional polymorphisms (rs1805502, rs890, rs1806201, and rs7301328) within GRIN2B were genotyped in all participants. The haplotype analysis found significant differences in the distribution of the G-T haplotype between the TRD and control groups (corrected P = 0.007), and the frequency of the G-T haplotype in TRD group was significantly higher than that in the controls (TRD/control ratio 0.31:0.21). Statistically significant differences in allele and genotype frequencies were detected between TRD and non-TRD groups for the rs1805502 polymorphism within GRIN2B. There was a significant allelic association between rs1805502 and TRD with an excess of the G allele in the TRD group, compared to non-TRD group (OR = 1.55, 95 % CI = 1.18-2.05, corrected P = 0.008).
CONCLUSIONS: These initial findings strengthen the hypothesis that GRIN2B not only confers susceptibility to TRD, but also plays a genetic predictor for TRD in MDD patients.

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Year:  2013        PMID: 24114429     DOI: 10.1007/s00213-013-3297-0

Source DB:  PubMed          Journal:  Psychopharmacology (Berl)        ISSN: 0033-3158            Impact factor:   4.530


  43 in total

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4.  [Mapping genes related to early onset major depressive disorder in Dagestan genetic isolates].

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Review 10.  Definition, assessment, and staging of treatment-resistant refractory major depression: a review of current concepts and methods.

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5.  Prefrontal Connectivity and Glutamate Transmission: Relevance to Depression Pathophysiology and Ketamine Treatment.

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Review 6.  Glutamate and its receptors in the pathophysiology and treatment of major depressive disorder.

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7.  Sex differences in glutamate receptor gene expression in major depression and suicide.

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10.  Metabotropic Glutamate Receptor 5 and Glutamate Involvement in Major Depressive Disorder: A Multimodal Imaging Study.

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