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Literature DB >> 24082058

Newly discovered components and actions of the renin-angiotensin system.

Robert M Carey¹.

Abstract

Entities: Gene

Mesh：

Substances：

Year: 2013 PMID： 24082058 DOI： 10.1161/HYPERTENSIONAHA.113.01111

Source DB: PubMed Journal: Hypertension ISSN： 0194-911X Impact factor: 10.190

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19 in total

1. Antenatal glucocorticoid treatment alters Na+ uptake in renal proximal tubule cells from adult offspring in a sex-specific manner.

Authors: Yixin Su; Jianli Bi; Victor M Pulgar; Jorge Figueroa; Mark Chappell; James C Rose
Journal: Am J Physiol Renal Physiol Date: 2015-04-01

Review 2. New components of the renin-angiotensin system: alamandine and the MAS-related G protein-coupled receptor D.

Authors: Gisele Maia Etelvino; Antônio Augusto Bastos Peluso; Robson Augusto Souza Santos
Journal: Curr Hypertens Rep Date: 2014-06 Impact factor: 5.369

Review 3. Angiotensin-(1-12): a chymase-mediated cellular angiotensin II substrate.

Authors: Sarfaraz Ahmad; Jasmina Varagic; Leanne Groban; Louis J Dell'Italia; Sayaka Nagata; Neal D Kon; Carlos M Ferrario
Journal: Curr Hypertens Rep Date: 2014-05 Impact factor: 5.369

4. Large arteriovenous malformation of the renal artery causing congestive heart failure.

Authors: M A Rana; B Sarwar; H Zabher; N I Akkus
Journal: Herz Date: 2014-03-28 Impact factor: 1.443

Review 5. Blood Pressure and the Renal Actions of AT₂ Receptors.

Authors: Robert M Carey
Journal: Curr Hypertens Rep Date: 2017-03 Impact factor: 5.369

Review 6. Update on angiotensin AT2 receptors.

Authors: Robert M Carey
Journal: Curr Opin Nephrol Hypertens Date: 2017-03 Impact factor: 2.894

10. TSH receptor signaling via cyclic AMP inhibits cell surface degradation and internalization of E-cadherin in pig thyroid epithelium.

Authors: F Larsson; H Fagman; M Nilsson
Journal: Cell Mol Life Sci Date: 2004-07 Impact factor: 9.261

Newly discovered components and actions of the renin-angiotensin system.

1. Antenatal glucocorticoid treatment alters Na+ uptake in renal proximal tubule cells from adult offspring in a sex-specific manner.

Review 2. New components of the renin-angiotensin system: alamandine and the MAS-related G protein-coupled receptor D.

Review 3. Angiotensin-(1-12): a chymase-mediated cellular angiotensin II substrate.

4. Large arteriovenous malformation of the renal artery causing congestive heart failure.

Review 5. Blood Pressure and the Renal Actions of AT₂ Receptors.

Review 6. Update on angiotensin AT2 receptors.

Review 7. Angiotensin II Signal Transduction: An Update on Mechanisms of Physiology and Pathophysiology.

8. Identification of a Primary Renal AT₂ Receptor Defect in Spontaneously Hypertensive Rats.

Review 9. The ACE2/Angiotensin-(1-7)/MAS Axis of the Renin-Angiotensin System: Focus on Angiotensin-(1-7).

10. TSH receptor signaling via cyclic AMP inhibits cell surface degradation and internalization of E-cadherin in pig thyroid epithelium.

Newly discovered components and actions of the renin-angiotensin system.

1. Antenatal glucocorticoid treatment alters Na+ uptake in renal proximal tubule cells from adult offspring in a sex-specific manner.

Review 2. New components of the renin-angiotensin system: alamandine and the MAS-related G protein-coupled receptor D.

Review 3. Angiotensin-(1-12): a chymase-mediated cellular angiotensin II substrate.

4. Large arteriovenous malformation of the renal artery causing congestive heart failure.

Review 5. Blood Pressure and the Renal Actions of AT2 Receptors.

Review 6. Update on angiotensin AT2 receptors.

Review 7. Angiotensin II Signal Transduction: An Update on Mechanisms of Physiology and Pathophysiology.

8. Identification of a Primary Renal AT2 Receptor Defect in Spontaneously Hypertensive Rats.

Review 9. The ACE2/Angiotensin-(1-7)/MAS Axis of the Renin-Angiotensin System: Focus on Angiotensin-(1-7).

10. TSH receptor signaling via cyclic AMP inhibits cell surface degradation and internalization of E-cadherin in pig thyroid epithelium.

Review 5. Blood Pressure and the Renal Actions of AT₂ Receptors.

8. Identification of a Primary Renal AT₂ Receptor Defect in Spontaneously Hypertensive Rats.