Literature DB >> 24050535

Increased arterial stiffness is independently associated with metabolic syndrome and damage index in systemic lupus erythematosus patients.

S Valero-Gonzalez1, R Castejon, C Jimenez-Ortiz, S Rosado, P Tutor-Ureta, J-A Vargas, M Yebra-Bango.   

Abstract

OBJECTIVES: We evaluated whether traditional or non-traditional cardiovascular (CV) risk factors and systemic lupus erythematosus (SLE)-related risk factors were associated with pathological arterial stiffness measured by pulse wave velocity (PWV) adjusted for patients' age and blood pressure.
METHOD: CV risk factors were measured in the 46 SLE female patients studied. Activity and organ damage were assessed by the SLE Disease Activity Index (SLEDAI) and the Systemic Lupus International Collaborative Clinics/American College of Rheumatology (SLICC/ACR) Damage Index, respectively. Other lupus-related parameters and information concerning treatment were recorded. Subclinical atherosclerosis was assessed by PWV calculated from pulse wave recording by Doppler, a non-invasive method to measure arterial stiffness. Multivariate logistic regression analysis was used to identify independent determinants of increased PWV.
RESULTS: PWV was categorized as normal or pathological arterial stiffness following the reference values adjusted by age and blood pressure recently published by the European Society of Cardiology. Pathological PWV was associated with CV risk factors including homocysteine (p = 0.01), high-sensitivity C-reactive protein (hs-CRP; p = 0.03), uric acid (p = 0.01), and metabolic syndrome (p = 0.007). With regard to SLE-specific risk factors, a significant association was found between PWV and SLICC/ACR score (p = 0.006). Multivariate analysis showed that increased PWV was independently associated with metabolic syndrome [odds ratio (OR) 6.6, 95% confidence interval (CI) 1.2-38, p = 0.03] and SLICC/ACR score (OR 1.5, 95% CI 1-2.32, p = 0.05).
CONCLUSIONS: We have found a close link between metabolic syndrome and SLICC/ACR score with increased aortic stiffness. These variables might be an indicator of subclinical atherosclerosis in SLE women without clinical evidence of atherosclerotic cardiovascular disease (CVD).

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Year:  2013        PMID: 24050535     DOI: 10.3109/03009742.2013.803150

Source DB:  PubMed          Journal:  Scand J Rheumatol        ISSN: 0300-9742            Impact factor:   3.641


  9 in total

1.  Peroxisome proliferator-activated receptor gamma agonists in the prevention and treatment of murine systemic lupus erythematosus.

Authors:  Tamar R Aprahamian; Ramon G Bonegio; Zachary Weitzner; Raffi Gharakhanian; Ian R Rifkin
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2.  Serum cystatin C is associated with kidney function but not with cardiovascular risk factors or subclinical atherosclerosis in patients with Systemic Lupus Erythematosus.

Authors:  Patricia Garcia-Garcia; Raquel Castejon; Pablo Tutor-Ureta; R A Silvestre; Susana Mellor-Pita; Carlos Jimenez-Ortiz; Miguel Yebra-Bango
Journal:  Clin Rheumatol       Date:  2017-09-15       Impact factor: 2.980

3.  Assessment of aortic stiffness among patients with systemic lupus erythematosus and rheumatoid arthritis by magnetic resonance imaging.

Authors:  Galia Karp; Arik Wolak; Yael Baumfeld; Nina Bar-Am; Victor Novack; Talya Wolak; Lior Fuchs; Aryeh Shalev; Ilan Shelef; Mahmoud Abu-Shakra
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4.  Dynamics of pulse wave velocity and vascular augmentation index in association with endothelial progenitor cells in SLE.

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8.  Overweight Is a Major Contributor to Atherosclerosis in Systemic Lupus Erythematosus Patients at Apparent Low Risk for Cardiovascular Disease: A Cross-Sectional Controlled Study.

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9.  Reduction in SLEDAI is associated with improved arterial stiffness in systemic lupus erythematosus.

Authors:  Tian Du; Haiyu Pang; Faming Ding; Yicong Ye; Mengtao Li; Xufei Yang; Yang Zhang; Xiaofeng Zeng; Shuyang Zhang
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  9 in total

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