| Literature DB >> 24045165 |
Alessandra Musella1, Georgia Mandolesi, Antonietta Gentile, Silvia Rossi, Valeria Studer, Caterina Motta, Helena Sepman, Diego Fresegna, Nabila Haji, Andrea Paolillo, Giuseppe Matarese, Diego Centonze.
Abstract
Alterations of glutamate-mediated synaptic transmission occur in both multiple sclerosis (MS) and experimental autoimmune encephalomyelitis (EAE), the animal model of MS. Here we investigated whether intracerebroventricular (Icv) administration of cladribine has effects on EAE. Icv infusion of cladribine reduced the clinical deficits of EAE mice and reversed EAE-induced enhancement of excitatory postsynaptic current (sEPSC) frequency, a neurophysiological measure of glutamatergic synaptopathy associated with central inflammation. Cladribine failed to interfere with EAE-induced microglial and astroglial activation, but blocked EAE synaptic alterations by interfering with interleukin-1β effects. Cladribine possesses neuroprotective properties in experimental MS that are independent of its peripheral immunosuppressant action.Entities:
Keywords: Cladribine; EAE; EPSC; IL-1β; Microglia; Striatum
Mesh:
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Year: 2013 PMID: 24045165 DOI: 10.1016/j.jneuroim.2013.08.009
Source DB: PubMed Journal: J Neuroimmunol ISSN: 0165-5728 Impact factor: 3.478