Literature DB >> 24023294

Effects of dietary calcium on Helicobacter pylori-induced gastritis in Mongolian gerbils.

Masaki Iimuro1, Shiro Nakamura, Tetsuo Arakawa, Keiji Wakabayashi, Michihiro Mutoh.   

Abstract

BACKGROUND AND AIMS: Helicobacter pylori (Hp) infection causes gastritis and is considered a gastric cancer risk factor. We have previously reported that codfish meal markedly enhanced Hp-induced gastritis in Mongolian gerbils. In the present study, we sought the responsible components in codfish meal.
MATERIALS AND METHODS: Codfish were divided into three parts (meat, viscera and 'other parts', including bone), and administered to Hp-infected gerbils. Subsequently, cod bone, sardine bone and prawn shell were tested, along with major calcium components, hydroxyapatite and calcium carbonate, in bone and shell, respectively.
RESULTS: 'Other parts' and cod bone enhanced Hp-induced gastritis, as was observed for whole codfish. Similarly, sardine bone and prawn shell, as well as 0.22-0.88% hydroxyapatite and calcium carbonate, enhanced gastritis. In contrast, administration of a higher dose of the calcium compounds exerted protective effects.
CONCLUSION: Intake of calcium compounds may contribute to enhancement of Hp-induced gastritis.

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Keywords:  Gastritis; Helicobacter pylori; Mongolian gerbils; calcium; fish meal

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Year:  2013        PMID: 24023294

Source DB:  PubMed          Journal:  Anticancer Res        ISSN: 0250-7005            Impact factor:   2.480


  1 in total

1.  NCX1 coupled with TRPC1 to promote gastric cancer via Ca2+/AKT/β-catenin pathway.

Authors:  Hanxing Wan; Nannan Gao; Wei Lu; Cheng Lu; Jun Chen; Yimin Wang; Hui Dong
Journal:  Oncogene       Date:  2022-07-26       Impact factor: 8.756

  1 in total

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