Literature DB >> 24010184

Decrease of brain zinc in experimental hepatic encephalopathy.

M Baraldi1, E Caselgrandi, P Borella, M L Zeneroli.   

Abstract

Since zinc is an important factor in membrane stability, we assayed the levels of zinc in several brain areas during the development of hepatic encephalopathy due to D-galactosamine-HCl in rat. We now report that zinc is significantly reduced in all tested brain areas. This finding seems to indicate an involvement of zinc in the changes of membrane properties that lead to alteration of GABA receptors in hepatic encephalopathy.

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Year:  1983        PMID： 24010184     DOI： 10.1016/0006-8993(83)91246-5

Source DB:  PubMed          Journal:  Brain Res        ISSN： 0006-8993            Impact factor:   3.252


  4 in total

1.  Histochemical demonstration of copper in normal rat brain and spinal cord. Evidence of localization in glial cells.

Authors:  P Szerdahelyi; P Kása
Journal:  Histochemistry       Date:  1986

Review 2.  Supersensitivity of GABA-A receptors in hepatic encephalopathy.

Authors:  M Baraldi
Journal:  Neurochem Res       Date:  1990-02       Impact factor: 3.996

Review 3.  Natural endogenous ligands for benzodiazepine receptors in hepatic encephalopathy.

Authors:  M Baraldi; R Avallone; L Corsi; I Venturini; C Baraldi; M L Zeneroli
Journal:  Metab Brain Dis       Date:  2008-12-11       Impact factor: 3.584

4.  Short-term oral zinc supplementation does not improve chronic hepatic encephalopathy. Results of a double-blind crossover trial.

Authors:  O Riggio; F Ariosto; M Merli; M Caschera; A Zullo; G Balducci; V Ziparo; G Pedretti; F Fiaccadori; E Bottari
Journal:  Dig Dis Sci       Date:  1991-09       Impact factor: 3.199
  4 in total

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