Literature DB >> 23963331

Granulocyte colony-stimulating factor in patients with acute ischemic stroke: results of the AX200 for Ischemic Stroke trial.

E Bernd Ringelstein1, Vincent Thijs, Bo Norrving, Angel Chamorro, Franz Aichner, Martin Grond, Jeff Saver, Rico Laage, Armin Schneider, Frank Rathgeb, Gerhard Vogt, Gabriele Charissé, Jochen B Fiebach, Stefan Schwab, Wolf R Schäbitz, Rainer Kollmar, Marc Fisher, Miroslav Brozman, David Skoloudik, Franz Gruber, Joaquin Serena Leal, Roland Veltkamp, Martin Köhrmann, Jörg Berrouschot.   

Abstract

BACKGROUND AND
PURPOSE: Granulocyte colony-stimulating factor (G-CSF; AX200; Filgrastim) is a stroke drug candidate with excellent preclinical evidence for efficacy. A previous phase IIa dose-escalation study suggested potential efficacy in humans. The present large phase IIb trial was powered to detect clinical efficacy in acute ischemic stroke patients.
METHODS: G-CSF (135 µg/kg body weight intravenous over 72 hours) was tested against placebo in 328 patients in a multinational, multicenter, randomized, and placebo-controlled trial (NCT00927836; www.clinicaltrial.gov). Main inclusion criteria were ≤9-hour time window after stroke onset, infarct localization in the middle cerebral artery territory, baseline National Institutes of Health Stroke Scale score range of 6 to 22, and baseline diffusion-weighted imaging lesion size ≥15 mL. Primary and secondary end points were the modified Rankin scale score and the National Institutes of Health Stroke Scale score at day 90, respectively. Data were analyzed using a prespecified model that adjusted for age, National Institutes of Health Stroke Scale score at baseline, and initial infarct volume (diffusion-weighted imaging).
RESULTS: G-CSF treatment failed to meet the primary and secondary end points of the trial. For additional end points such as mortality, Barthel index, or infarct size at day 30, G-CSF did not show efficacy either. There was, however, a trend for reduced infarct growth in the G-CSF group. G-CSF showed the expected peripheral pharmacokinetic and pharmacodynamic profiles, with a strong increase in leukocytes and monocytes. In parallel, the cytokine profile showed a significant decrease of interleukin-1.
CONCLUSIONS: G-CSF, a novel and promising drug candidate with a comprehensive preclinical and clinical package, did not provide any significant benefit with respect to either clinical outcome or imaging biomarkers. CLINICAL TRIAL REGISTRATION URL: http://www.clinicaltrials.gov. Unique identifier: NCT00927836.

Entities:  

Keywords:  G-CSF; MRI; clinical trial; hematopoietic growth factor; leukocytes; phase II; stroke

Mesh:

Substances:

Year:  2013        PMID: 23963331     DOI: 10.1161/STROKEAHA.113.001531

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


  44 in total

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2.  Association Between Time From Stroke Onset and Fluid-Attenuated Inversion Recovery Lesion Intensity Is Modified by Status of Collateral Circulation.

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3.  Association between the perfusion/diffusion and diffusion/FLAIR mismatch: data from the AXIS2 trial.

Authors:  Anke Wouters; Patrick Dupont; Erich B Ringelstein; Bo Norrving; Angel Chamorro; Martin Grond; Rico Laage; Armin Schneider; Guido Wilms; Götz Thomalla; Robin Lemmens; Vincent N Thijs
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Review 10.  Inflammatory Disequilibrium in Stroke.

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Journal:  Circ Res       Date:  2016-06-24       Impact factor: 17.367

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