Literature DB >> 23897582

Prediagnostic levels of serum one-carbon metabolites and risk of hepatocellular carcinoma.

Lesley M Butler1, Erland Arning, Renwei Wang, Teodoro Bottiglieri, Sugantha Govindarajan, Yu-Tang Gao, Jian-Min Yuan.   

Abstract

BACKGROUND: Rats fed diets deficient in choline develop hepatocellular carcinoma. Tumor DNA from these animals is characteristically hypomethylated, suggesting that disruption of the one-carbon metabolism pathway is an underlying mechanism for hepatocarcinogenesis. Prospective studies in humans on circulating choline and other one-carbon metabolites and hepatocellular carcinoma risk have been lacking.
METHODS: We prospectively examined the association between prediagnostic serum concentrations of one-carbon metabolites including betaine, choline, cystathionine, homocysteine, methionine, 5-methyltetrahydrofolate (5-MTHF), pyridoxal-5-phosphate (PLP, the bioactive form of vitamin B6) and S-adenosylmethionine (SAM), and risk of developing hepatocellular carcinoma based on a nested case-control study of 297 incident cases and 631 matched controls from a cohort of 18,244 men in Shanghai, China. Logistic regression methods were used to calculate ORs and 95% confidence intervals (CI) adjusted for established risk factors for hepatocellular carcinoma.
RESULTS: Serum choline and PLP were associated with statistically significant reduced risk of hepatocellular carcinoma, whereas serum cystathionine, methionine, and SAM were associated with increased hepatocellular carcinoma risk (all Ptrend < 0.05). The inverse associations for hepatocellular carcinoma risk with choline and PLP remained statistically significant after adjusting for all potential confounders. The multivariate-adjusted ORs (95% CIs) for the highest versus lowest quintiles of serum choline and PLP were 0.35 (0.16-0.78; P = 0.010) and 0.44 (0.25-0.78; P = 0.005), respectively. There were no associations for hepatocellular carcinoma risk with 5-MTHF, betaine, or homocysteine.
CONCLUSION: The inverse associations between choline and vitamin B6 and the risk of hepatocellular carcinoma development are novel and warrant further investigation. IMPACT: Identifying new modifiable factors for hepatocellular carcinoma prevention is warranted.

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Year:  2013        PMID: 23897582      PMCID: PMC3928816          DOI: 10.1158/1055-9965.EPI-13-0497

Source DB:  PubMed          Journal:  Cancer Epidemiol Biomarkers Prev        ISSN: 1055-9965            Impact factor:   4.254


  47 in total

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2.  In vivo regulation by glutathione of methionine adenosyltransferase S-nitrosylation in rat liver.

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Authors:  F Donato; P Boffetta; M Puoti
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4.  Deletion of betaine-homocysteine S-methyltransferase in mice perturbs choline and 1-carbon metabolism, resulting in fatty liver and hepatocellular carcinomas.

Authors:  Ya-Wen Teng; Mihai G Mehedint; Timothy A Garrow; Steven H Zeisel
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Authors:  G S Qian; R K Ross; M C Yu; J M Yuan; Y T Gao; B E Henderson; G N Wogan; J D Groopman
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3.  Global Level of Plasma DNA Methylation is Associated with Overall Survival in Patients with Hepatocellular Carcinoma.

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Authors:  Lesley M Butler; Joyce Yongxu Huang; Renwei Wang; Mao-Jun Lee; Chung S Yang; Yu-Tang Gao; Jian-Min Yuan
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9.  Higher dietary intakes of choline and betaine are associated with a lower risk of primary liver cancer: a case-control study.

Authors:  Rui-Fen Zhou; Xiao-Lin Chen; Zhong-Guo Zhou; Yao-Jun Zhang; Qiu-Ye Lan; Gong-Cheng Liao; Yu-Ming Chen; Hui-Lian Zhu
Journal:  Sci Rep       Date:  2017-04-06       Impact factor: 4.379

10.  One-carbon metabolism-related micronutrients intake and risk for hepatocellular carcinoma: A prospective cohort study.

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