Literature DB >> 23887230

Physical exercise induces specific adaptations resulting in reduced organ injury and mortality during severe polymicrobial sepsis.

Maik Sossdorf1, Jacqueline Fischer, Stefan Meyer, Katja Dahlke, Bianka Wissuwa, Carolin Seidel, Andrea Schrepper, Clemens L Bockmeyer, Amelie Lupp, Sophie Neugebauer, Diana Schmerler, Jürgen Rödel, Ralf A Claus, Gordon P Otto.   

Abstract

OBJECTIVES: High physical activity levels are associated with wide-ranging health benefits, disease prevention, and longevity. In the present study, we examined the impact of regular physical exercise on the severity of organ injury and survival probability, as well as characteristics of the systemic immune and metabolic response during severe polymicrobial sepsis.
DESIGN: Animal study.
SETTING: University laboratory.
SUBJECTS: Male C57BL/6N mice.
INTERVENTIONS: Mice were trained for 6 weeks by treadmill and voluntary wheel running or housed normally. Polymicrobial sepsis in mice was induced by injection of fecal slurry. Subsequently, mice were randomized into the following groups: healthy controls, 6 hours postsepsis, and 24 hours postsepsis.
MEASUREMENTS AND MAIN RESULTS: Blood and organ samples were collected and investigated by measuring clinical chemistry variables, cytokines, plasma metabolites, and bacterial clearance. Organ morphology and damage were characterized by histological staining. Physical exercise improved survival and the ability of bacterial clearance in blood and organs. The release of pro- and anti-inflammatory cytokines, including interleukin-6 and interleukin-10, was diminished in trained compared to untrained mice during sepsis. The sepsis-associated acute kidney tubular damage was less pronounced in pretrained animals. By metabolic profiling and regression analysis, we detected lysophosphatidylcholine 14:0, tryptophan, as well as pimelylcarnitine linked with levels of neutrophil gelatinase-associated lipocalin representing acute tubular injury (corrected R=0.910; p<0.001). We identified plasma lysophosphatidylcholine 16:0, lysophosphatidylcholine 17:0, and lysophosphatidylcholine 18:0 as significant metabolites discriminating between trained and untrained mice during sepsis.
CONCLUSIONS: Regular physical exercise reduces sepsis-associated acute kidney injury and death. As a specific mechanism of exercise-induced adaptation, we identified various lysophosphatidylcholines that might function as surrogate for improved outcome in sepsis.

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Year:  2013        PMID: 23887230     DOI: 10.1097/CCM.0b013e31828a2ae3

Source DB:  PubMed          Journal:  Crit Care Med        ISSN: 0090-3493            Impact factor:   7.598


  11 in total

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Review 10.  Exercise as a therapeutic option for acute kidney injury: mechanisms and considerations for the design of future clinical studies.

Authors:  Anam Asad; James O Burton; Daniel S March
Journal:  BMC Nephrol       Date:  2020-10-23       Impact factor: 2.388

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