Literature DB >> 23857399

A phase II study of the histone deacetylase inhibitor panobinostat (LBH589) in pretreated patients with small-cell lung cancer.

Filippo de Marinis1, Akin Atmaca, Marcello Tiseo, Libero Giuffreda, Antonio Rossi, Vittorio Gebbia, Chiara D'Antonio, Laura Dal Zotto, Salah-Eddin Al-Batran, Silvia Marsoni, Martin Wolf.   

Abstract

BACKGROUND: In vitro data suggest that panobinostat (LBH589), a pan-deacetylase inhibitor, may add therapeutic benefit in the treatment of small-cell lung cancer (SCLC) with regression of tumors.
METHODS: This multicenter, nonrandomized phase 2 trial was designed to evaluate antitumor activity of LBH589 in patients with previously treated SCLC. Patients received LBH589 administered intravenously at a dose of 20 mg/mq (days 1-8) every 21 days.
RESULTS: A total of 21 patients with extensive- or limited-stage SCLC were enrolled. Patients received a median of two cycles (range, 1-6). LBH589 was well tolerated, and the most common toxicities were grade 1 to 2 gastrointestinal disorders (nausea 38%, diarrhea 24%, vomiting 19%), grade 1 to 2 thrombocytopenia (14.3%). Of 19 patients evaluable for efficacy, two cases showed shrinkages more than 30% at first assessment, with time to progression of 14 and 21 weeks, respectively, and there were three long disease stabilizations of 12, 10, and 13 weeks. The study was prematurely closed because of a lack of activity.
CONCLUSION: This is the first report of a pan-deacetylase inhibitor inducing tumor shrinkage and sustained stable disease in SCLC. We believe that although the trial was prematurely discontinued, modest clinical activity of LBH589 combined with a favorable safety profile in pretreated SCLC patients was observed, which warrants further exploration of the potential contribution of LBH589 in other trials.

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Year:  2013        PMID: 23857399     DOI: 10.1097/JTO.0b013e318293d88c

Source DB:  PubMed          Journal:  J Thorac Oncol        ISSN: 1556-0864            Impact factor:   15.609


  14 in total

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7.  Molecular Mechanism of the Cell Death Induced by the Histone Deacetylase Pan Inhibitor LBH589 (Panobinostat) in Wilms Tumor Cells.

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Review 8.  Histone deacetylase inhibitors in clinical studies as templates for new anticancer agents.

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10.  Comparison of the antifibrotic effects of the pan-histone deacetylase-inhibitor panobinostat versus the IPF-drug pirfenidone in fibroblasts from patients with idiopathic pulmonary fibrosis.

Authors:  Martina Korfei; Daniel Stelmaszek; BreAnne MacKenzie; Sylwia Skwarna; Shashipavan Chillappagari; Anna C Bach; Clemens Ruppert; Shigeki Saito; Poornima Mahavadi; Walter Klepetko; Ludger Fink; Werner Seeger; Joseph A Lasky; Soni S Pullamsetti; Oliver H Krämer; Andreas Guenther
Journal:  PLoS One       Date:  2018-11-27       Impact factor: 3.240

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