Literature DB >> 23828159

Association of abstinence-induced alterations in working memory function and COMT genotype in smokers.

Rebecca L Ashare1, Jeffrey N Valdez, Kosha Ruparel, Benjamin Albelda, Ryan D Hopson, John R Keefe, James Loughead, Caryn Lerman.   

Abstract

RATIONALE: The common methionine (met) for valine (val) at codon 158 (val(158)met) polymorphism in the catechol-O-methyltransferase (COMT) gene has been associated with nicotine dependence, alterations in executive cognitive function, and abstinence-induced working memory deficits in smokers.
OBJECTIVES: We sought to replicate the association of the COMT val allele with abstinence-induced alterations in working memory-related activity in task-positive (executive control) and task-negative (default mode network) regions.
METHODS: Forty smokers (20 val/val and 20 met/met) performed an N-back task while undergoing blood oxygen level-dependent (BOLD) functional magnetic resonance imaging (fMRI) on two separate occasions: following 72 h of confirmed abstinence and during smoking as usual. An independent sample of 48 smokers who completed the identical N-back task during fMRI in smoking vs. abstinence for another study was used as a validation sample.
RESULTS: Contrary to expectations, genotype by session interactions on BOLD signal in executive control regions (dorsolateral prefrontal cortex and dorsal cingulate/medial prefrontal cortex) revealed significant abstinence-induced reductions in the met/met group, but not the val/val group. Results also revealed that val/val smokers may exhibit less suppression of activation in task-negative regions such as the posterior cingulate cortex during abstinence (vs. smoking). These patterns were confirmed in the validation sample and in the whole-brain analysis, though the regions differed from the a priori regions of interest (ROIs) (e.g., precuneus, insula).
CONCLUSIONS: The COMT val(158)met polymorphism was associated with abstinence-related working memory deficits in two independent samples of smokers. However, inconsistencies compared to prior findings and across methods (ROI vs. whole-brain analysis) highlight the challenges inherent in reproducing results of imaging genetic studies in addiction.

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Year:  2013        PMID: 23828159      PMCID: PMC3840089          DOI: 10.1007/s00213-013-3197-3

Source DB:  PubMed          Journal:  Psychopharmacology (Berl)        ISSN: 0033-3158            Impact factor:   4.530


  62 in total

1.  COMT Val158Met polymorphism in relation to activation and de-activation in the prefrontal cortex: A study in patients with schizophrenia and healthy subjects.

Authors:  E Pomarol-Clotet; M Fatjó-Vilas; P J McKenna; G C Monté; S Sarró; J Ortiz-Gil; C Aguirre; J J Gomar; A Guerrero; R Landin; A Capdevila; L Fañanás; R Salvador
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Review 2.  Are genetic variants of COMT associated with addiction?

Authors:  Anne Emilia Tammimäki; Pekka T Männistö
Journal:  Pharmacogenet Genomics       Date:  2010-12       Impact factor: 2.089

Review 3.  The catechol-O-methyltransferase gene: its regulation and polymorphisms.

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Journal:  Int Rev Neurobiol       Date:  2010       Impact factor: 3.230

4.  Nicotine replacement in abstinent smokers improves cognitive withdrawal symptoms with modulation of resting brain network dynamics.

Authors:  David M Cole; Christian F Beckmann; Christopher J Long; Paul M Matthews; Michael J Durcan; John D Beaver
Journal:  Neuroimage       Date:  2010-05-02       Impact factor: 6.556

5.  Effects of the alpha4beta2 partial agonist varenicline on brain activity and working memory in abstinent smokers.

Authors:  James Loughead; Riju Ray; E Paul Wileyto; Kosha Ruparel; Paul Sanborn; Steven Siegel; Ruben C Gur; Caryn Lerman
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Review 6.  Cognitive enhancers in the treatment of substance use disorders: clinical evidence.

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7.  The effects of the COMT Val108/158Met polymorphism on BOLD activation during working memory, planning, and response inhibition: a role for the posterior cingulate cortex?

Authors:  Paul R A Stokes; Rebecca A Rhodes; Paul M Grasby; Mitul A Mehta
Journal:  Neuropsychopharmacology       Date:  2010-12-08       Impact factor: 7.853

8.  Working memory deficits predict short-term smoking resumption following brief abstinence.

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Review 9.  Cognitive enhancement as a pharmacotherapy target for stimulant addiction.

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10.  Neural substrates of pleiotropic action of genetic variation in COMT: a meta-analysis.

Authors:  D Mier; P Kirsch; A Meyer-Lindenberg
Journal:  Mol Psychiatry       Date:  2009-05-05       Impact factor: 15.992

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  5 in total

Review 1.  Genetic studies of alcohol dependence in the context of the addiction cycle.

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Journal:  Neuropharmacology       Date:  2017-01-22       Impact factor: 5.250

2.  COMT polymorphism modulates the resting-state EEG alpha oscillatory response to acute nicotine in male non-smokers.

Authors:  H Bowers; D Smith; S de la Salle; J Choueiry; D Impey; T Philippe; H Dort; A Millar; M Daigle; P R Albert; A Beaudoin; V Knott
Journal:  Genes Brain Behav       Date:  2015-07-15       Impact factor: 3.449

Review 3.  Optimizing treatments for nicotine dependence by increasing cognitive performance during withdrawal.

Authors:  Rebecca L Ashare; Heath D Schmidt
Journal:  Expert Opin Drug Discov       Date:  2014-04-07       Impact factor: 6.098

4.  Withdrawal-Related Changes in Delay Discounting Predict Short-Term Smoking Abstinence.

Authors:  Rickie Miglin; Joseph W Kable; Maureen E Bowers; Rebecca L Ashare
Journal:  Nicotine Tob Res       Date:  2017-06-01       Impact factor: 4.244

Review 5.  Cognitive Effects of Nicotine: Recent Progress.

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