Literature DB >> 23809475

Plasma cytokine levels and cytokine gene polymorphisms in Mexican patients during the influenza pandemic A(H1N1)pdm09.

Joel Martinez-Ocaña1, Angélica Olivo-Diaz, Tobías Salazar-Dominguez, Jesús Reyes-Gordillo, Cynthia Tapia-Aquino, Fernando Martínez-Hernández, María Eugenia Manjarrez, Marco Antonio-Martinez, Araceli Contreras-Molina, Rafael Figueroa-Moreno, Rafael Valdez-Vazquez, Simón Kawa-Karasik, Patricia Rodríguez-Zulueta, Ana Flisser, Pablo Maravilla, Mirza Romero-Valdovinos.   

Abstract

BACKGROUND: In Mexico, the initial severe cases of the 2009 influenza pandemic virus A (H1N1) [A(H1N1)pdm09] were detected in early March. The immune mechanisms associated with the severe pneumonia caused by infection with this new virus have not been completely elucidated. Polymorphisms in interleukin genes have previously been associated with susceptibility to infectious diseases due to their influence on cytokine production.
OBJECTIVES: The present case-control study was performed to compare several immunologic and genetic parameters of patients and controls during the initial phase of the pandemic. STUDY
DESIGN: Sixty-five patients who were hospitalized due to infection with the influenza A(H1N1)pdm09 virus and 46 healthy controls were studied. A hemagglutination inhibition assay (HIA) was performed to measure anti-influenza antibody titers in these subjects. Protein levels of the cytokines interleukin (IL)-4, IL-6, IL-8, IL-10, tumor necrosis factor-α (TNFα), interferon gamma (IFNγ), transforming growth factor beta (TGFβ)1 and TGFβ2 were quantified in plasma. Single nucleotide polymorphisms in IL6, IL10 and TNFα were also assessed.
RESULTS: Influenza patients had lower antibody titers and produced significantly higher levels of IL-6, IL-10 and TNFα than healthy controls. The frequencies of the TNFα -308G, IL-10 -592C and IL-10 -1082A alleles and the IL10 -1082(A/A) genotype were associated with susceptibility to severe disease, while the haplotypes TNFα AG and IL-10 GTA and GCA were associated with protection from severe disease [P=0.016, OR (CI)=0.11 (0.01-0.96); P=0.0187, OR (CI)=0.34 (0.13-0.85); P=0.013, OR (CI)=0.39 (0.18-0.83)].
CONCLUSIONS: This study demonstrates that the influenza A(H1N1)pdm09 patients and healthy controls have different profiles of immune parameters and that there is an association between IL-10 and TNFα polymorphisms and the outcome of this disease.
Copyright © 2013 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  A(H1N1)pdm09; Cytokine plasma levels; Cytokine polymorphism; HIA; Hemagglutination Inhibition assay; IL-10; Influenza; Pandemic A/H1N1

Mesh:

Substances:

Year:  2013        PMID: 23809475     DOI: 10.1016/j.jcv.2013.05.013

Source DB:  PubMed          Journal:  J Clin Virol        ISSN: 1386-6532            Impact factor:   3.168


  17 in total

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Authors:  Erick J Rendón-Ramirez; Alejandro Ortiz-Stern; Corazon Martinez-Mejia; Mario C Salinas-Carmona; Adrian Rendon; Viviana L Mata-Tijerina; Adrian G Rosas-Taraco
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Authors:  Tamara V Smelaya; Olesya B Belopolskaya; Svetlana V Smirnova; Artem N Kuzovlev; Viktor V Moroz; Arkadiy M Golubev; Noel A Pabalan; Lyubov E Salnikova
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Review 7.  Interindividual immunogenic variants: Susceptibility to coronavirus, respiratory syncytial virus and influenza virus.

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8.  TNF, IL6, and IL1B Polymorphisms Are Associated with Severe Influenza A (H1N1) Virus Infection in the Mexican Population.

Authors:  Román Alejandro García-Ramírez; Alejandra Ramírez-Venegas; Roger Quintana-Carrillo; Ángel Eduardo Camarena; Ramcés Falfán-Valencia; Juan Manuel Mejía-Aranguré
Journal:  PLoS One       Date:  2015-12-14       Impact factor: 3.240

9.  Cytokine Profiles of Severe Influenza Virus-Related Complications in Children.

Authors:  Andrew Fiore-Gartland; Angela Panoskaltsis-Mortari; Anna A Agan; Anushay J Mistry; Paul G Thomas; Michael A Matthay; Tomer Hertz; Adrienne G Randolph
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10.  Anti-IL-6 receptor antibody treatment for severe COVID-19 and the potential implication of IL-6 gene polymorphisms in novel coronavirus pneumonia.

Authors:  Zulvikar Syambani Ulhaq; Gita Vita Soraya
Journal:  Med Clin (Barc)       Date:  2020-07-10       Impact factor: 1.725

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