Literature DB >> 23739141

In HIV/hepatitis C virus co-infected patients, higher 25-hydroxyvitamin D concentrations were not related to hepatitis C virus treatment responses but were associated with ritonavir use.

Andrea D Branch1, Minhee Kang, Kimberly Hollabaugh, Christina M Wyatt, Raymond T Chung, Marshall J Glesby.   

Abstract

BACKGROUND: Among patients with hepatitis C virus (HCV) monoinfection, 25-hydroxyvitamin D [25(OH)D] concentrations are positively associated with a response to peg-interferon/ribavirin. Data on the relation between 25(OH)D concentrations and HCV treatment response in HIV-infected patients are limited.
OBJECTIVE: The objective was to determine whether baseline 25(OH)D concentrations predict virologic response in HIV/HCV co-infected patients and to examine variables associated with 25(OH)D concentrations ≥30 ng/mL.
DESIGN: Data and samples from 144 HCV genotype 1, treatment-naive patients from a completed HCV treatment trial were examined in this retrospective study. Early virologic response (EVR) was defined as ≥2 log10 reduction in HCV RNA and/or HCV RNA <600 IU/mL at week 12 of peg-interferon/ribavirin treatment. Baseline 25(OH)D was measured by liquid chromatography/tandem mass spectrometry.
RESULTS: Compared with the non-EVR control group (n = 68), the EVR group (n = 76) was younger, had fewer cirrhotic subjects, had a higher proportion with the IL28B CC genotype, had a higher albumin concentration, and had a lower HCV viral load at baseline (P ≤ 0.05). The difference in baseline 25(OH)D concentrations between EVR and non-EVR patients was not statistically significant (median: 25 ng/mL compared with 20 ng/mL; P = 0.23). Similar results were found for sustained virologic response (SVR). In multivariable analysis, white and Hispanic race-ethnicity (OR: 6.26; 95% CI: 2.47, 15.88; P = 0.0001) and ritonavir use (OR: 2.68; 95% CI: 1.08, 6.65; P = 0.033) were associated with higher 25(OH)D concentrations (≥30 ng/mL).
CONCLUSION: Baseline 25(OH)D concentrations did not predict EVR or SVR. Because ritonavir impairs the conversion of 25(OH)D to the active metabolite, utilization of 25(OH)D may have been impaired in subjects taking ritonavir. This trial was registered at www.clinicaltrials.gov as NCT00078403.

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Year:  2013        PMID: 23739141      PMCID: PMC3712551          DOI: 10.3945/ajcn.112.048785

Source DB:  PubMed          Journal:  Am J Clin Nutr        ISSN: 0002-9165            Impact factor:   7.045


  59 in total

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Review 2.  Hepatitis C virus infection as an opportunistic disease in persons infected with human immunodeficiency virus.

Authors:  M S Sulkowski; E E Mast; L B Seeff; D L Thomas
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3.  Vitamin D supplementation improves sustained virologic response in chronic hepatitis C (genotype 1)-naïve patients.

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Review 4.  Classical and emerging roles of vitamin D in hepatitis C virus infection.

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6.  Low vitamin D levels are associated with impaired virologic response to PEGIFN + RBV therapy in HIV-hepatitis C virus coinfected patients.

Authors:  Mattias Mandorfer; Thomas Reiberger; Berit A Payer; Arnulf Ferlitsch; Florian Breitenecker; Maximilian C Aichelburg; Barbara Obermayer-Pietsch; Armin Rieger; Michael Trauner; Markus Peck-Radosavljevic
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7.  Vitamin D levels and IL28B polymorphisms are related to rapid virological response to standard of care in genotype 1 chronic hepatitis C.

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8.  Combined effect of 25-OH vitamin D plasma levels and genetic vitamin D receptor (NR 1I1) variants on fibrosis progression rate in HCV patients.

Authors:  Katharina Baur; Joachim C Mertens; Johannes Schmitt; Rika Iwata; Bruno Stieger; Jyrki J Eloranta; Pascal Frei; Felix Stickel; Michael T Dill; Burkhardt Seifert; Heike A Bischoff Ferrari; Arnold von Eckardstein; Pierre-Yves Bochud; Beat Müllhaupt; Andreas Geier
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9.  Increasing mortality due to end-stage liver disease in patients with human immunodeficiency virus infection.

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10.  Vitamin D status does not predict sustained virologic response or fibrosis stage in chronic hepatitis C genotype 1 infection.

Authors:  Matthew T Kitson; Gregory J Dore; Jacob George; Peter Button; Geoffrey W McCaughan; Darrell H G Crawford; William Sievert; Martin D Weltman; Wendy S Cheng; Stuart K Roberts
Journal:  J Hepatol       Date:  2012-11-23       Impact factor: 25.083

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  2 in total

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Review 2.  Vitamin D Deficiency in HIV Infection: Not Only a Bone Disorder.

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  2 in total

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