Literature DB >> 23512795

Trk B signaling in dopamine 1 receptor neurons regulates food intake and body weight.

Brittany L Mason1, Mary Kay Lobo, Luis F Parada, Michael Lutter.   

Abstract

OBJECTIVE: Loss of BDNF-TrkB signaling results in obesity in both humans and mice; however, the neural circuit that mediates this effect is unknown. The role of TrkB signaling in dopamine-1 receptor expressing neurons in body weight regulation was tested.
METHODS: Mice with a floxed allele of the TrkB gene were paired with mice expressing Cre-recombinase under control of the D1 promoter to conditionally knock out expression of TrkB receptors from D1-neurons.
RESULTS: Deletion of TrkB receptors from D1 neurons results in obesity in chow fed mice due to increased feed efficiency. In contrast, loss of Trk B signaling in D1 neurons induced hyperphagia and hyperglycemia in mice maintained on high fat diet.
CONCLUSIONS: These findings indicate TrkB signaling in D1 neurons regulates body weight by distinct mechanisms for chow and high fat diet and may be important for defending the body against the development of obesity and obesity-related disorders.
Copyright © 2013 The Obesity Society.

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Year:  2013        PMID: 23512795      PMCID: PMC3742719          DOI: 10.1002/oby.20382

Source DB:  PubMed          Journal:  Obesity (Silver Spring)        ISSN: 1930-7381            Impact factor:   5.002


  13 in total

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  4 in total

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2.  Novel and ultra-rare damaging variants in neuropeptide signaling are associated with disordered eating behaviors.

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4.  BDNF influences neural cue-reactivity to food stimuli and food craving in obesity.

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  4 in total

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