Literature DB >> 23500641

Drosophila melanogaster as an emerging translational model of human nephrolithiasis.

Joe Miller1, Thomas Chi, Pankaj Kapahi, Arnold J Kahn, Man Su Kim, Taku Hirata, Michael F Romero, Julian A T Dow, Marshall L Stoller.   

Abstract

PURPOSE: The limitations imposed by human clinical studies and mammalian models of nephrolithiasis have hampered the development of effective medical treatments and preventive measures for decades. The simple but elegant Drosophila melanogaster is emerging as a powerful translational model of human disease, including nephrolithiasis. It may provide important information essential to our understanding of stone formation. We present the current state of research using D. melanogaster as a model of human nephrolithiasis.
MATERIALS AND METHODS: We comprehensively reviewed the English language literature using PubMed®. When necessary, authoritative texts on relevant subtopics were consulted.
RESULTS: The genetic composition, anatomical structure and physiological function of Drosophila malpighian tubules are remarkably similar to those of the human nephron. The direct effects of dietary manipulation, environmental alteration and genetic variation on stone formation can be observed and quantified in a matter of days. Several Drosophila models of human nephrolithiasis have been developed, including genetically linked and environmentally induced stones. A model of calcium oxalate stone formation is among the most recent fly models of human nephrolithiasis.
CONCLUSIONS: The ability to readily manipulate and quantify stone formation in D. melanogaster models of human nephrolithiasis presents the urological community with a unique opportunity to increase our understanding of this enigmatic disease.
Copyright © 2013 American Urological Association Education and Research, Inc. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  ATPase; CT; Drosophila melanogaster; UAS; XDH; adenosine triphosphatase; animal; computerized tomography; disease models; kidney; malpighian tubules; nephrolithiasis; upstream activation sequence; xanthine dehydrogenase

Mesh:

Year:  2013        PMID: 23500641      PMCID: PMC3842186          DOI: 10.1016/j.juro.2013.03.010

Source DB:  PubMed          Journal:  J Urol        ISSN: 0022-5347            Impact factor:   7.450


  34 in total

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3.  Mutation of human molybdenum cofactor sulfurase gene is responsible for classical xanthinuria type II.

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Journal:  Am J Physiol Renal Physiol       Date:  2012-09-19

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Authors:  M E Thompson; M R Lewin-Smith; V F Kalasinsky; K M Pizzolato; M L Fleetwood; M R McElhaney; T O Johnson
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2.  Antiurolithic effects of medicinal plants: results of in vivo studies in rat models of calcium oxalate nephrolithiasis-a systematic review.

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8.  Sulfate and thiosulfate inhibit oxalate transport via a dPrestin (Slc26a6)-dependent mechanism in an insect model of calcium oxalate nephrolithiasis.

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10.  A Drosophila model identifies a critical role for zinc in mineralization for kidney stone disease.

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