Literature DB >> 23499266

Everolimus in hormone receptor-positive advanced breast cancer: targeting receptor-based mechanisms of resistance.

Mikhail I Shtivelband1.   

Abstract

Although patients with hormone receptor (HR)-positive breast cancer are successfully treated with endocrine therapy, many tumors go on to develop resistance to these agents. Studies have determined that mechanisms of resistance to endocrine therapy are quite complex and can involve a multitude of signal transduction pathways, either through direct association with the estrogen receptor or through cross-talk with other pathways. Preclinical studies have suggested the therapeutic importance of the mammalian target of rapamycin (mTOR) pathway and that inhibiting this pathway may restore sensitivity to endocrine therapy. The oral mTOR inhibitor everolimus has been extensively studied for breast cancer. Clinical studies suggest that everolimus in combination with endocrine therapy improves progression-free survival and is well tolerated. A combined approach, targeting both mTOR signal transduction and the HR pathways, promises to take clinical research in a new direction for the treatment of HR-positive advanced breast cancer.
Copyright © 2013 Elsevier Ltd. All rights reserved.

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Year:  2013        PMID: 23499266     DOI: 10.1016/j.breast.2013.02.003

Source DB:  PubMed          Journal:  Breast        ISSN: 0960-9776            Impact factor:   4.380


  9 in total

1.  Mammalian target of rapamycin (mTOR) inhibitors and combined chemotherapy in breast cancer: a meta-analysis of randomized controlled trials.

Authors:  Longwei Qiao; Yuting Liang; Ranim R Mira; Yaojuan Lu; Junxia Gu; Qiping Zheng
Journal:  Int J Clin Exp Med       Date:  2014-10-15

Review 2.  Management of patients with hormone receptor-positive breast cancer with visceral disease: challenges and treatment options.

Authors:  Wael A Harb
Journal:  Cancer Manag Res       Date:  2015-01-21       Impact factor: 3.989

3.  Cyclin-dependent kinase 11(p110) (CDK11(p110)) is crucial for human breast cancer cell proliferation and growth.

Authors:  Yubing Zhou; Chao Han; Duolu Li; Zujiang Yu; Fengmei Li; Feng Li; Qi An; Huili Bai; Xiaojian Zhang; Zhenfeng Duan; Quancheng Kan
Journal:  Sci Rep       Date:  2015-05-20       Impact factor: 4.379

Review 4.  Biomarkers of Everolimus Sensitivity in Hormone Receptor-Positive Breast Cancer.

Authors:  Zongbi Yi; Fei Ma
Journal:  J Breast Cancer       Date:  2017-12-19       Impact factor: 3.588

Review 5.  Molecular Signaling of Progesterone, Growth Hormone, Wnt, and HER in Mammary Glands of Dogs, Rodents, and Humans: New Treatment Target Identification.

Authors:  Elpetra P M Timmermans-Sprang; Ana Gracanin; Jan A Mol
Journal:  Front Vet Sci       Date:  2017-04-13

6.  Everolimus inhibits breast cancer cell growth through PI3K/AKT/mTOR signaling pathway.

Authors:  Liyan Du; Xiaomei Li; Linhong Zhen; Weiling Chen; Lingguang Mu; Yang Zhang; Ailin Song
Journal:  Mol Med Rep       Date:  2018-03-16       Impact factor: 2.952

Review 7.  Autophagy Modulators: Mechanistic Aspects and Drug Delivery Systems.

Authors:  Shima Tavakol; Milad Ashrafizadeh; Shuo Deng; Maryam Azarian; Asghar Abdoli; Mahsa Motavaf; Delaram Poormoghadam; Hashem Khanbabaei; Elham Ghasemipour Afshar; Ali Mandegary; Abbas Pardakhty; Celestial T Yap; Reza Mohammadinejad; Alan Prem Kumar
Journal:  Biomolecules       Date:  2019-09-25

8.  Focal Adhesion Kinase Provides a Collateral Vulnerability That Can Be Leveraged to Improve mTORC1 Inhibitor Efficacy.

Authors:  Leslie Cuellar-Vite; Kristen L Weber-Bonk; Fadi W Abdul-Karim; Christine N Booth; Ruth A Keri
Journal:  Cancers (Basel)       Date:  2022-07-11       Impact factor: 6.575

Review 9.  Signal Transduction Pathways in Breast Cancer: The Important Role of PI3K/Akt/mTOR.

Authors:  Miguel A Ortega; Oscar Fraile-Martínez; Ángel Asúnsolo; Julia Buján; Natalio García-Honduvilla; Santiago Coca
Journal:  J Oncol       Date:  2020-03-09       Impact factor: 4.375

  9 in total

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