Literature DB >> 23496221

CRAC inhibitors: identification and potential.

Isabella Derler1, Reinhard Fritsch, Rainer Schindl, Christoph Romanin.   

Abstract

BACKGROUND: Ca(2+) release-activated Ca(2+) (CRAC) channels, a subfamily of store-operated channels, play an essential role in various diseases such as immune disorders and allergic responses.
OBJECTIVE: The successful treatment of these diseases requires the identification of specific inhibitors. So far, a variety of chemical compounds blocking CRAC have been identified; however, they have all turned out to be less specific. Recently two proteins, STIM1 and ORAI1, have been identified as the essential components that fully reconstitute CRAC currents with a similar biophysical fingerprint.
METHOD: These two proteins and their activation process represent direct targets for the application of specific CRAC inhibitors. RESULTS/
CONCLUSION: For drug development, fluorescence microscopy adaptable for high-throughput screening will provide a powerful assay to mechanistically identify potential CRAC inhibitors that act on various stages within the STIM1/ORAI1 activation pathway visualized by fluorescent-tagged proteins.

Entities:  

Year:  2008        PMID: 23496221     DOI: 10.1517/17460441.3.7.787

Source DB:  PubMed          Journal:  Expert Opin Drug Discov        ISSN: 1746-0441            Impact factor:   6.098


  19 in total

1.  S1P activates store-operated calcium entry via receptor- and non-receptor-mediated pathways in vascular smooth muscle cells.

Authors:  Kristen Park Hopson; Jessica Truelove; Jerold Chun; Yumei Wang; Christian Waeber
Journal:  Am J Physiol Cell Physiol       Date:  2011-01-26       Impact factor: 4.249

Review 2.  Store-Operated Calcium Channels.

Authors:  Murali Prakriya; Richard S Lewis
Journal:  Physiol Rev       Date:  2015-10       Impact factor: 37.312

3.  Benzoxazine Derivatives As CRAC Modulators.

Authors:  Benjamin Blass
Journal:  ACS Med Chem Lett       Date:  2013-10-10       Impact factor: 4.345

4.  Overexpression of Orai1 and STIM1 proteins alters regulation of store-operated Ca2+ entry by endogenous mediators.

Authors:  Tomasz Gwozdz; Joanna Dutko-Gwozdz; Claudia Schafer; Victoria M Bolotina
Journal:  J Biol Chem       Date:  2012-05-01       Impact factor: 5.157

5.  Anchoring protein AKAP79-mediated PKA phosphorylation of STIM1 determines selective activation of the ARC channel, a store-independent Orai channel.

Authors:  Jill L Thompson; Trevor J Shuttleworth
Journal:  J Physiol       Date:  2015-01-07       Impact factor: 5.182

Review 6.  STIM and Orai: the long-awaited constituents of store-operated calcium entry.

Authors:  Péter Várnai; László Hunyady; Tamas Balla
Journal:  Trends Pharmacol Sci       Date:  2009-01-31       Impact factor: 14.819

7.  Suppression of store-operated Ca2+ entry regulated by silencing Orai1 inhibits C6 glioma cell motility via decreasing Pyk2 activity and promoting focal adhesion.

Authors:  Meng Zhu; Bingke Lv; Wenjing Ge; Zhenwen Cui; Kai Zhao; Yugong Feng; Xuejun Yang
Journal:  Cell Cycle       Date:  2020-12-03       Impact factor: 4.534

8.  Visualisation and identification of the interaction between STIM1s in resting cells.

Authors:  Jun He; Tao Yu; Jingying Pan; He Li
Journal:  PLoS One       Date:  2012-03-16       Impact factor: 3.240

9.  The boron-oxygen core of borinate esters is responsible for the store-operated calcium entry potentiation ability.

Authors:  Olivier Dellis; Pierre Mercier; Christine Chomienne
Journal:  BMC Pharmacol       Date:  2011-01-26

10.  The action of selective CRAC channel blockers is affected by the Orai pore geometry.

Authors:  Isabella Derler; Rainer Schindl; Reinhard Fritsch; Peter Heftberger; Maria Christine Riedl; Malcolm Begg; David House; Christoph Romanin
Journal:  Cell Calcium       Date:  2012-12-05       Impact factor: 6.817

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