Literature DB >> 23494723

The changing clinical course of multiple sclerosis: a matter of gray matter.

Massimiliano Calabrese1, Chiara Romualdi, Valentina Poretto, Alice Favaretto, Aldo Morra, Francesca Rinaldi, Paola Perini, Paolo Gallo.   

Abstract

OBJECTIVE: Clinical and neuroimaging parameters predictive of the changing clinical course of multiple sclerosis (MS) from relapsing-remitting to secondary progressive have not been clarified yet. We specifically designed a prospective 5-year longitudinal study aimed at assessing demographic, clinical, and magnetic resonance imaging (MRI) parameters that could predict the changing clinical course of MS.
METHODS: At study entry and after 5 years, clinical and MRI (ie, gray matter and white matter lesions, including spinal cord lesions, and global and regional cortical thinning) parameters were assessed in a training set of 334 consecutive relapsing-remitting MS patients and in an independent validation set of 84 relapsing-remitting MS patients.
RESULTS: Sixty-six (19.7%) relapsing-remitting MS patients changed their clinical course during the study and entered into the secondary progressive phase. Age (p = 0.001, odds ratio [OR] = 1.2), cortical lesion volume (p < 0.001, OR = 1.7), and cerebellar cortical volume (p < 0.001, OR = 0.2) at study entry were found to predict the changing clinical course. The model including only these 3 variables correctly identified 252 of 268 (94.0%) patients who maintained the relapsing-remitting course and 58 of 66 (87.8%) patients who became secondary progressive (cross-validated error rate = 7.2%). When applied on the validation set, the model obtained a similar error rate (8.4%).
INTERPRETATION: A prediction model based on age, cortical lesion load, and cerebellar cortical volume suitably explains the probability of relapsing-remitting MS patients evolving into the progressive phase. Gray matter damage appears to play a pivotal role in determining the changing clinical course of MS.
Copyright © 2013 American Neurological Association.

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Mesh:

Year:  2013        PMID: 23494723     DOI: 10.1002/ana.23882

Source DB:  PubMed          Journal:  Ann Neurol        ISSN: 0364-5134            Impact factor:   10.422


  17 in total

1.  FLAIR2: A Combination of FLAIR and T2 for Improved MS Lesion Detection.

Authors:  V Wiggermann; E Hernández-Torres; A Traboulsee; D K B Li; A Rauscher
Journal:  AJNR Am J Neuroradiol       Date:  2015-10-08       Impact factor: 3.825

2.  Improved Visualization of Cortical Lesions in Multiple Sclerosis Using 7T MP2RAGE.

Authors:  E S Beck; P Sati; V Sethi; T Kober; B Dewey; P Bhargava; G Nair; I C Cortese; D S Reich
Journal:  AJNR Am J Neuroradiol       Date:  2018-02-08       Impact factor: 3.825

3.  Modeling the relationship among gray matter atrophy, abnormalities in connecting white matter, and cognitive performance in early multiple sclerosis.

Authors:  A F Kuceyeski; W Vargas; M Dayan; E Monohan; C Blackwell; A Raj; K Fujimoto; S A Gauthier
Journal:  AJNR Am J Neuroradiol       Date:  2014-11-20       Impact factor: 3.825

4.  Diffusion kurtosis imaging probes cortical alterations and white matter pathology following cuprizone induced demyelination and spontaneous remyelination.

Authors:  C Guglielmetti; J Veraart; E Roelant; Z Mai; J Daans; J Van Audekerke; M Naeyaert; G Vanhoutte; R Delgado Y Palacios; J Praet; E Fieremans; P Ponsaerts; J Sijbers; A Van der Linden; M Verhoye
Journal:  Neuroimage       Date:  2015-10-23       Impact factor: 6.556

5.  The effect of fingolimod on focal and diffuse grey matter damage in active MS patients.

Authors:  Albulena Bajrami; Marco Pitteri; Marco Castellaro; Francesca Pizzini; Chiara Romualdi; Stefania Montemezzi; Salvatore Monaco; Massimiliano Calabrese
Journal:  J Neurol       Date:  2018-06-25       Impact factor: 4.849

6.  Disease-modifying drugs reduce cortical lesion accumulation and atrophy progression in relapsing-remitting multiple sclerosis: results from a 48-month extension study.

Authors:  Francesca Rinaldi; Paola Perini; Matteo Atzori; Alice Favaretto; Dario Seppi; Paolo Gallo
Journal:  Mult Scler Int       Date:  2015-02-23

7.  Cortical pathology in multiple sclerosis detected by the T1/T2-weighted ratio from routine magnetic resonance imaging.

Authors:  Ruthger Righart; Viola Biberacher; Laura E Jonkman; Roel Klaver; Paul Schmidt; Dorothea Buck; Achim Berthele; Jan S Kirschke; Claus Zimmer; Bernhard Hemmer; Jeroen J G Geurts; Mark Mühlau
Journal:  Ann Neurol       Date:  2017-09-16       Impact factor: 10.422

8.  Temporal and spatial evolution of grey matter atrophy in primary progressive multiple sclerosis.

Authors:  Arman Eshaghi; Benedetta Bodini; Gerard R Ridgway; Daniel García-Lorenzo; Daniel J Tozer; Mohammad Ali Sahraian; Alan J Thompson; Olga Ciccarelli
Journal:  Neuroimage       Date:  2013-10-04       Impact factor: 6.556

Review 9.  Clinical, MRI, and CSF markers of disability progression in multiple sclerosis.

Authors:  Alberto Gajofatto; Massimiliano Calabrese; Maria Donata Benedetti; Salvatore Monaco
Journal:  Dis Markers       Date:  2013-11-10       Impact factor: 3.434

10.  Complement is activated in progressive multiple sclerosis cortical grey matter lesions.

Authors:  Lewis M Watkins; James W Neal; Sam Loveless; Iliana Michailidou; Valeria Ramaglia; Mark I Rees; Richard Reynolds; Neil P Robertson; B Paul Morgan; Owain W Howell
Journal:  J Neuroinflammation       Date:  2016-06-22       Impact factor: 8.322

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