Literature DB >> 23453027

Assessing the contribution of HRPT2 to the pathogenesis of jaw fibrous dysplasia, ossifying fibroma, and osteosarcoma.

Ana Carolina de Mesquita Netto1, Ricardo Santiago Gomez, Marina Gonçalves Diniz, Thiago Fonseca-Silva, Kelma Campos, Luiz De Marco, Román Carlos, Carolina Cavaliéri Gomes.   

Abstract

OBJECTIVE: To investigate HRPT2 in jaw ossifying fibroma (OF), fibrous dysplasia (FD), and osteosarcoma (OS). STUDY
DESIGN: We combined microsatellite loss of heterozygosity (LOH), HRPT2 sequence alterations at the mRNA level by reverse-transcription polymerase chain reaction (PCR), cDNA sequencing, and quantitative PCR (qPCR) and immunohistochemistry (IHC) in a total of 19 OF, 15 FD, and 9 OS. Because HRPT2 (parafibromin) interacts with cyclin D1, we investigated cyclin D1 expression with the use of qPCR and IHC.
RESULTS: LOH was detected in 3/5 FD, 6/9 OF, and 2/2 OS heterozygous samples. LOH was not associated with decreased mRNA levels or HRPT2 protein expression except for 1 OF which harbored an inactivating mutation. However, this tumor did not display altered transcription or protein levels of HRPT2 nor cyclin compared with the other OF.
CONCLUSIONS: The contribution of HRPT2 inactivation to the pathogenesis of OF, FD, and OS is marginal at best and may be limited to progression rather than tumor initiation.
Copyright © 2013 Elsevier Inc. All rights reserved.

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Year:  2013        PMID: 23453027     DOI: 10.1016/j.oooo.2012.11.015

Source DB:  PubMed          Journal:  Oral Surg Oral Med Oral Pathol Oral Radiol


  7 in total

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Authors:  Saurabh R Nagar; Neha Mittal; Swapnil U Rane; Munita Bal; Asawari Patil; Suman Kumar Ankathi; Shivakumar Thiagarajan
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7.  CDC73 gene mutations in sporadic ossifying fibroma of the jaws.

Authors:  Yan Chen; Da-Yan Hu; Ting-Ting Wang; Ran Zhang; Qing Dong; Zhi-Xiu Xu; Lin Wang; Tie-Jun Li
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  7 in total

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