Literature DB >> 23375034

Carvacrol suppresses the expression of inflammatory marker genes in D-galactosamine-hepatotoxic rats.

Balakrishnan Aristatile1, Abdullah H Al-Assaf, Kodukkur Viswanathan Pugalendi.   

Abstract

OBJECTIVE: To unravel the mechanism of anti-inflammatory activity of carvacrol in D-galactosamine (D-GalN)-induced hepatotoxic rats.
METHODS: The mRNA and protein expression levels of tumor necrosis factor-alpha (TNF-α), interleukin-6 (IL-6), inducible nitric oxide synthase (iNOS), cyclooxygenase-2 (COX-2) and nuclear factor kappa-B (NF-κB) were assayed by semi-quantitative reverse transcriptase polymerase chain reaction (RTPCR) and western blot analysis.
RESULTS: We found that the mRNA and protein expressions of TNF-α, IL-6, iNOS, COX-2 and NF-κB were significantly up-regulated in D-galactosamine induced hepatotoxic rats and treatment with carvacrol significantly down-regulated the expressions of these genes showing the mechanism behind the anti-inflammatory activity of carvacrol.
CONCLUSIONS: All above results reveal that the carvacrol well known anti-inflammatory activities in D-galactosamine induced hepatotoxic rats.
Copyright © 2013 Hainan Medical College. Published by Elsevier B.V. All rights reserved.

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Year:  2013        PMID: 23375034     DOI: 10.1016/S1995-7645(13)60024-3

Source DB:  PubMed          Journal:  Asian Pac J Trop Med        ISSN: 1995-7645            Impact factor:   1.226


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