Literature DB >> 23333450

Essential fatty acids deficiency promotes lipogenic gene expression and hepatic steatosis through the liver X receptor.

Simon Ducheix1, Alexandra Montagner, Arnaud Polizzi, Frédéric Lasserre, Alice Marmugi, Justine Bertrand-Michel, Normand Podechard, Talal Al Saati, Maud Chétiveaux, Silvère Baron, Jérôme Boué, Gilles Dietrich, Laila Mselli-Lakhal, Philippe Costet, Jean-Marc A Lobaccaro, Thierry Pineau, Vassilia Theodorou, Catherine Postic, Pascal G P Martin, Hervé Guillou.   

Abstract

BACKGROUND & AIMS: Nutrients influence non-alcoholic fatty liver disease. Essential fatty acids deficiency promotes various syndromes, including hepatic steatosis, through increased de novo lipogenesis. The mechanisms underlying such increased lipogenic response remain unidentified.
METHODS: We used wild type mice and mice lacking Liver X Receptors to perform a nutrigenomic study that aimed at examining the role of these transcription factors.
RESULTS: We showed that, in the absence of Liver X Receptors, essential fatty acids deficiency does not promote steatosis. Consistent with this, Liver X Receptors are required for the elevated expression of genes involved in lipogenesis in response to essential fatty acids deficiency.
CONCLUSIONS: This work identifies, for the first time, the central role of Liver X Receptors in steatosis induced by essential fatty acids deficiency.
Copyright © 2013 European Association for the Study of the Liver. Published by Elsevier B.V. All rights reserved.

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Year:  2013        PMID: 23333450     DOI: 10.1016/j.jhep.2013.01.006

Source DB:  PubMed          Journal:  J Hepatol        ISSN: 0168-8278            Impact factor:   25.083


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