Literature DB >> 23314960

Intraamniotic lipopolysaccharide exposure changes cell populations and structure of the ovine fetal thymus.

Elke Kuypers1, Tim G A M Wolfs, Jennifer J P Collins, Reint K Jellema, John P Newnham, Matthew W Kemp, Suhas G Kallapur, Alan H Jobe, Boris W Kramer.   

Abstract

RATIONALE: Chorioamnionitis induces preterm delivery and acute involution of the fetal thymus which is associated with postnatal inflammatory disorders. We studied the immune response, cell composition, and architecture of the fetal thymus following intraamniotic lipopolysaccharide (LPS) exposure.
METHODS: Time-mated ewes received an intraamniotic injection of LPS 5, 12, or 24 hours or 2, 4, 8, or 15 days before delivery at 125 days gestational age (term = 150 days).
RESULTS: The LPS exposure resulted in decreased blood lymphocytes within 5 hours and decreased thymic corticomedullary ratio within 24 hours. Thymic interleukin 6 (IL6) and IL17 messenger RNA (mRNA) increased 5-fold 24 hours post-LPS exposure. Increased toll-like receptor 4 (TLR4) mRNA and nuclear factor κB positive cells at 24 hours after LPS delivery demonstrated acute thymic activation. Both TLR4 and IL1 mRNA increased by 5-fold and the number of Foxp3-positive cells (Foxp3+ cells) decreased 15 days after exposure.
CONCLUSION: Intraamniotic LPS exposure caused a proinflammatory response, involution, and a persistent depletion of thymic Foxp3+ cells indicating disturbance of the fetal immune homeostasis.

Entities:  

Keywords:  fetal inflammatory response syndrome; immune modulation; prematurity; thymic involution

Mesh:

Substances:

Year:  2013        PMID: 23314960      PMCID: PMC3702021          DOI: 10.1177/1933719112472742

Source DB:  PubMed          Journal:  Reprod Sci        ISSN: 1933-7191            Impact factor:   3.060


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