Literature DB >> 23305873

Akt2 expression is associated with good long-term prognosis in oestrogen receptor positive breast cancer.

Helena Fohlin1, Gizeh Pérez-Tenorio, Tommy Fornander, Lambert Skoog, Bo Nordenskjöld, John Carstensen, Olle Stål.   

Abstract

INTRODUCTION: Akt is a signalling modulator for many cellular processes, including metabolism, cell proliferation, cell survival and cell growth. Three isoforms of Akt have been identified, but only a few studies have concerned the isoform-specific roles in the prognosis of breast cancer patients. The aim of this study was to investigate the prognostic value of v-akt murine thymoma viral oncogene homologue 1 (Akt1) and v-akt murine thymoma viral oncogene homologue 2 (Akt2) in oestrogen receptor positive (ER+) and oestrogen receptor negative (ER-) breast cancer with long-term follow-up.
MATERIAL AND METHODS: The expression of Akt in tumour tissue was analysed with immunohistochemistry in a cohort of 272 postmenopausal patients with stage II breast cancer. The median follow-up time was 19 years. Hazard ratios (HRs) and 95% confidence intervals (CIs) were estimated using the Cox's proportional hazards model.
RESULTS: The risk of distant recurrence was reduced for patients with ER+ tumours expressing Akt2 compared to patients with no Akt2 expression (HR=0.49, 95% CI 0.29-0.82, p=0.007). When adjusting for important clinical tumour characteristics and treatment, Akt2 was still an independent prognostic factor (HR=0.38, 95% CI 0.21-0.68, p=0.001) and the association remained long-term. The prognostic value of Akt2 increased with higher oestrogen receptor levels from no effect among patients with ER- tumours to 68% risk reduction for the group with high ER-levels (P for trend=0.042). Akt1 showed no significant prognostic information.
CONCLUSION: Our results indicate that Akt2 expression is associated with a lower distant recurrence rate for patients with ER+ tumours and that this association remains long-term. The prognostic value of Akt2 increases with higher oestrogen receptor expression, motivating further mechanistic studies on the role of Akt2 in ER+ breast cancer.
Copyright © 2012 Elsevier Ltd. All rights reserved.

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Year:  2013        PMID: 23305873     DOI: 10.1016/j.ejca.2012.12.006

Source DB:  PubMed          Journal:  Eur J Cancer        ISSN: 0959-8049            Impact factor:   9.162


  5 in total

1.  MiR-615 inhibited cell proliferation and cell cycle of human breast cancer cells by suppressing of AKT2 expression.

Authors:  Yang Bai; Jingyan Li; Jie Li; Yonghong Liu; Bo Zhang
Journal:  Int J Clin Exp Med       Date:  2015-03-15

2.  Tumor-treating fields induce autophagy by blocking the Akt2/miR29b axis in glioblastoma cells.

Authors:  Eun Ho Kim; Yunhui Jo; Sei Sai; Mung-Jin Park; Jeong-Yub Kim; Jin Su Kim; Yeon-Joo Lee; Jae-Min Cho; Seo-Young Kwak; Jeong-Hwa Baek; Youn Kyoung Jeong; Jie-Young Song; Myonggeun Yoon; Sang-Gu Hwang
Journal:  Oncogene       Date:  2019-08-02       Impact factor: 9.867

3.  Long noncoding RNA H19 contributes to gallbladder cancer cell proliferation by modulated miR-194-5p targeting AKT2.

Authors:  Shou-Hua Wang; Xiao-Cai Wu; Ming-Di Zhang; Ming-Zhe Weng; Di Zhou; Zhi-Wei Quan
Journal:  Tumour Biol       Date:  2016-01-23

4.  AKT1 and AKT2 isoforms play distinct roles during breast cancer progression through the regulation of specific downstream proteins.

Authors:  Marina Riggio; María C Perrone; María L Polo; María J Rodriguez; María May; Martín Abba; Claudia Lanari; Virginia Novaro
Journal:  Sci Rep       Date:  2017-03-13       Impact factor: 4.379

Review 5.  Distinct functions of AKT isoforms in breast cancer: a comprehensive review.

Authors:  Nico Hinz; Manfred Jücker
Journal:  Cell Commun Signal       Date:  2019-11-21       Impact factor: 5.712

  5 in total

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