Literature DB >> 23265768

Azacitidine in fludarabine-refractory chronic lymphocytic leukemia: a phase II study.

Asifa Malik1, Mahran Shoukier, Guillermo Garcia-Manero, William Wierda, Jorge Cortes, Susan Bickel, Michael J Keating, Zeev Estrov.   

Abstract

BACKGROUND: Treatment of fludarabine-refractory disease in patients with chronic lymphocytic leukemia (CLL) remains a challenge. Because a recent genome-wide methylation analysis of CLL cells suggested that demethylation therapy might be beneficial in CLL, we conducted a phase II trial with the hypomethylating agent azacitidine in patients with recurrent fludarabine-refractory CLL. PATIENTS AND METHODS: Nine patients with recurrent fludarabine-refractory Rai stage IV CLL (median age, 74 years; range, 49-81 years) were enrolled. Azacitidine (75 mg/m(2)) was administered by subcutaneous injection daily for 7 consecutive days every 3 to 8 weeks, and the data were analyzed at a median follow-up of 9 months (range 3-47 months).
RESULTS: The trial was prematurely discontinued because of lack of response and slow accrual. The number of cycles administered ranged from 1 to 6. Three patients received 1 cycle, 3 patients received 2 cycles, and the remaining 3 patients received 4, 5, or 6 cycles. Side effects included grade 2 or 3 infectious episodes (resulting from immunosuppression and drug-induced neutropenia), diarrhea, rash, vomiting, anemia, and thrombocytopenia. One patient experienced reduction of hepatosplenomegaly and a substantial increase in platelet count after 4 cycles of therapy. However this response did not qualify as a partial response according to the National Cancer Institute International Workshop on CLL (NCI-IWCLL) criteria. At a median follow-up of 9 months after the start of azacitidine treatment, 3 patients (33%) who went on to receive other treatments were alive.
CONCLUSIONS: Although no partial or complete responses occurred in these heavily pretreated patients, the encouraging response in 1 of these patients may warrant further studies to investigate the effects of azacitidine in CLL.
Copyright © 2013 Elsevier Inc. All rights reserved.

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Year:  2012        PMID: 23265768      PMCID: PMC3860181          DOI: 10.1016/j.clml.2012.11.009

Source DB:  PubMed          Journal:  Clin Lymphoma Myeloma Leuk        ISSN: 2152-2669


  13 in total

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2.  Genome-wide DNA methylation profiling of chronic lymphocytic leukemia allows identification of epigenetically repressed molecular pathways with clinical impact.

Authors:  Wei-Gang Tong; William G Wierda; E Lin; Shao-Qing Kuang; B Nebiyou Bekele; Zeev Estrov; Yue Wei; Hui Yang; Michael J Keating; Guillermo Garcia-Manero
Journal:  Epigenetics       Date:  2010-08-16       Impact factor: 4.528

3.  Addition of rituximab to fludarabine and cyclophosphamide in patients with chronic lymphocytic leukaemia: a randomised, open-label, phase 3 trial.

Authors:  M Hallek; K Fischer; G Fingerle-Rowson; A M Fink; R Busch; J Mayer; M Hensel; G Hopfinger; G Hess; U von Grünhagen; M Bergmann; J Catalano; P L Zinzani; F Caligaris-Cappio; J F Seymour; A Berrebi; U Jäger; B Cazin; M Trneny; A Westermann; C M Wendtner; B F Eichhorst; P Staib; A Bühler; D Winkler; T Zenz; S Böttcher; M Ritgen; M Mendila; M Kneba; H Döhner; S Stilgenbauer
Journal:  Lancet       Date:  2010-10-02       Impact factor: 79.321

4.  Cyclophosphamide, fludarabine, alemtuzumab, and rituximab as salvage therapy for heavily pretreated patients with chronic lymphocytic leukemia.

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6.  Ofatumumab is active in patients with fludarabine-refractory CLL irrespective of prior rituximab: results from the phase 2 international study.

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7.  RNA-dependent inhibition of ribonucleotide reductase is a major pathway for 5-azacytidine activity in acute myeloid leukemia.

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8.  Fludarabine plus alemtuzumab versus fludarabine alone in patients with previously treated chronic lymphocytic leukaemia: a randomised phase 3 trial.

Authors:  Thomas Elter; Liana Gercheva-Kyuchukova; Halyna Pylylpenko; Tadesuz Robak; Branimir Jaksic; Grigoriy Rekhtman; Sławomira Kyrcz-Krzemień; Mykola Vatutin; Jingyang Wu; Cynthia Sirard; Michael Hallek; Andreas Engert
Journal:  Lancet Oncol       Date:  2011-10-10       Impact factor: 41.316

9.  Phase I study of epigenetic modulation with 5-azacytidine and valproic acid in patients with advanced cancers.

Authors:  Fadi Braiteh; Andres O Soriano; Guillermo Garcia-Manero; David Hong; Marcella M Johnson; Leandro De Padua Silva; Hui Yang; Stefanie Alexander; Johannes Wolff; Razelle Kurzrock
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10.  Hematologic response to three alternative dosing schedules of azacitidine in patients with myelodysplastic syndromes.

Authors:  Roger M Lyons; Thomas M Cosgriff; Sanjiv S Modi; Robert H Gersh; John D Hainsworth; Allen L Cohn; Heidi J McIntyre; Indra J Fernando; Jay T Backstrom; C L Beach
Journal:  J Clin Oncol       Date:  2009-03-02       Impact factor: 44.544

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1.  Interaction between myelomonocytic and lymphoid cells in a patient with acute myelomonocytic leukemia and chronic lymphocytic leukemia.

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2.  A pilot clinical trial of the cytidine deaminase inhibitor tetrahydrouridine combined with decitabine to target DNMT1 in advanced, chemorefractory pancreatic cancer.

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3.  A pilot clinical trial of oral tetrahydrouridine/decitabine for noncytotoxic epigenetic therapy of chemoresistant lymphoid malignancies.

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4.  Identification of therapeutic candidates for chronic lymphocytic leukemia from a library of approved drugs.

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5.  Genetic methylation and lymphoid malignancies: biomarkers of tumor progression and targeted therapy.

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Journal:  Biomark Res       Date:  2013-08-14

Review 6.  Emerging epigenetic-modulating therapies in lymphoma.

Authors:  David Sermer; Laura Pasqualucci; Hans-Guido Wendel; Ari Melnick; Anas Younes
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