Literature DB >> 23239154

Critical illness myopathy and GLUT4: significance of insulin and muscle contraction.

Steffen Weber-Carstens1, Joanna Schneider, Tobias Wollersheim, Anke Assmann, Jeffrey Bierbrauer, Andreas Marg, Hadi Al Hasani, Alexandra Chadt, Katrin Wenzel, Susanne Koch, Jens Fielitz, Christian Kleber, Katharina Faust, Knut Mai, Claudia D Spies, Friedrich C Luft, Michael Boschmann, Joachim Spranger, Simone Spuler.   

Abstract

RATIONALE: Critical illness myopathy (CIM) has no known cause and no treatment. Immobilization and impaired glucose metabolism are implicated.
OBJECTIVES: We assessed signal transduction in skeletal muscle of patients at risk for CIM. We also investigated the effects of evoked muscle contraction.
METHODS: In a prospective observational and interventional pilot study, we screened 874 mechanically ventilated patients with a sepsis-related organ-failure assessment score greater than or equal to 8 for 3 consecutive days in the first 5 days of intensive care unit stay. Thirty patients at risk for CIM underwent euglycemic-hyperinsulinemic clamp, muscle microdialysis studies, and muscle biopsies. Control subjects were healthy. In five additional patients at risk for CIM, we performed corresponding analyses after 12-day, daily, unilateral electrical muscle stimulation with the contralateral leg as control.
MEASUREMENTS AND MAIN RESULTS: We performed successive muscle biopsies and assessed systemic insulin sensitivity and signal transduction pathways of glucose utilization at the mRNA and protein level and glucose transporter-4 (GLUT4) localization in skeletal muscle tissue. Skeletal muscle GLUT4 was trapped at perinuclear spaces, most pronounced in patients with CIM, but resided at the sarcolemma in control subjects. Glucose metabolism was not stimulated during euglycemic-hyperinsulinergic clamp. Insulin signal transduction was competent up to p-Akt activation; however, p-adenosine monophosphate-activated protein kinase (p-AMPK) was not detectable in CIM muscle. Electrical muscle stimulation increased p-AMPK, repositioned GLUT4, locally improved glucose metabolism, and prevented type-2 fiber atrophy.
CONCLUSIONS: Insufficient GLUT4 translocation results in decreased glucose supply in patients with CIM. Failed AMPK activation is involved. Evoked muscle contraction may prevent muscle-specific AMPK failure, restore GLUT4 disposition, and diminish protein breakdown. Clinical trial registered with http://www.controlled-trials.com (registration number ISRCTN77569430).

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Year:  2012        PMID: 23239154     DOI: 10.1164/rccm.201209-1649OC

Source DB:  PubMed          Journal:  Am J Respir Crit Care Med        ISSN: 1073-449X            Impact factor:   21.405


  41 in total

1.  [Acute muscle loss in critically ill patients].

Authors:  S Weber-Carstens
Journal:  Med Klin Intensivmed Notfmed       Date:  2014-04-26       Impact factor: 0.840

2.  Defective fatty acid oxidation in mice with muscle-specific acyl-CoA synthetase 1 deficiency increases amino acid use and impairs muscle function.

Authors:  Liyang Zhao; Florencia Pascual; Lawrence Bacudio; Amanda L Suchanek; Pamela A Young; Lei O Li; Sarah A Martin; Joao-Paulo Camporez; Rachel J Perry; Gerald I Shulman; Eric L Klett; Rosalind A Coleman
Journal:  J Biol Chem       Date:  2019-04-11       Impact factor: 5.157

Review 3.  The ICM research agenda on intensive care unit-acquired weakness.

Authors:  Nicola Latronico; Margaret Herridge; Ramona O Hopkins; Derek Angus; Nicholas Hart; Greet Hermans; Theodore Iwashyna; Yaseen Arabi; Giuseppe Citerio; E. Wesley Ely; Jesse Hall; Sangeeta Mehta; Kathleen Puntillo; Johannes Van den Hoeven; Hannah Wunsch; Deborah Cook; Claudia Dos Santos; Gordon Rubenfeld; Jean-Louis Vincent; Greet Van den Berghe; Elie Azoulay; Dale M Needham
Journal:  Intensive Care Med       Date:  2017-03-13       Impact factor: 17.440

Review 4.  The Sick and the Weak: Neuropathies/Myopathies in the Critically Ill.

Authors:  O Friedrich; M B Reid; G Van den Berghe; I Vanhorebeek; G Hermans; M M Rich; L Larsson
Journal:  Physiol Rev       Date:  2015-07       Impact factor: 37.312

5.  Rapid changes in the microvascular circulation of skeletal muscle impair insulin delivery during sepsis.

Authors:  Nicholas A Mignemi; P Mason McClatchey; Kameron V Kilchrist; Ian M Williams; Bryan A Millis; Kristen E Syring; Craig L Duvall; David H Wasserman; Owen P McGuinness
Journal:  Am J Physiol Endocrinol Metab       Date:  2019-03-12       Impact factor: 4.310

6.  Predictive value for weakness and 1-year mortality of screening electrophysiology tests in the ICU.

Authors:  Greet Hermans; Helena Van Mechelen; Frans Bruyninckx; Tine Vanhullebusch; Beatrix Clerckx; Philippe Meersseman; Yves Debaveye; Michael P Casaer; Alexander Wilmer; Pieter J Wouters; Ilse Vanhorebeek; Rik Gosselink; Greet Van den Berghe
Journal:  Intensive Care Med       Date:  2015-08-13       Impact factor: 17.440

7.  GTS-21 attenuates loss of body mass, muscle mass, and function in rats having systemic inflammation with and without disuse atrophy.

Authors:  Stefan J Schaller; Michio Nagashima; Martin Schönfelder; Tomoki Sasakawa; Fabian Schulz; Mohammed A S Khan; William R Kem; Gerhard Schneider; Jürgen Schlegel; Heidrun Lewald; Manfred Blobner; J A Jeevendra Martyn
Journal:  Pflugers Arch       Date:  2018-07-13       Impact factor: 3.657

8.  Malfolded protein structure and proteostasis in lung diseases.

Authors:  William E Balch; Jacob I Sznajder; Scott Budinger; Daniel Finley; Aaron D Laposky; Ana Maria Cuervo; Ivor J Benjamin; Esther Barreiro; Richard I Morimoto; Lisa Postow; Allan M Weissman; Dorothy Gail; Susan Banks-Schlegel; Thomas Croxton; Weiniu Gan
Journal:  Am J Respir Crit Care Med       Date:  2014-01-01       Impact factor: 21.405

Review 9.  [Critical illness polyneuropathy and myopathy as neurological complications of sepsis].

Authors:  R Kollmar
Journal:  Nervenarzt       Date:  2016-03       Impact factor: 1.214

10.  Dynamics of myosin degradation in intensive care unit-acquired weakness during severe critical illness.

Authors:  Tobias Wollersheim; Janine Woehlecke; Martin Krebs; Jida Hamati; Doerte Lodka; Anja Luther-Schroeder; Claudia Langhans; Kurt Haas; Theresa Radtke; Christian Kleber; Claudia Spies; Siegfried Labeit; Markus Schuelke; Simone Spuler; Joachim Spranger; Steffen Weber-Carstens; Jens Fielitz
Journal:  Intensive Care Med       Date:  2014-02-15       Impact factor: 17.440

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