Literature DB >> 23230322

Antitumor activity of cell-permeable RUNX3 protein in gastric cancer cells.

Junghee Lim1, Tam Duong, Nga Do, Phuong Do, Jaetaek Kim, Hyuncheol Kim, Wael El-Rifai, H Earl Ruley, Daewoong Jo.   

Abstract

PURPOSE: Gastric cancer is a leading cause of cancer death worldwide. Limited therapeutic options highlight the need to understand the molecular changes responsible for the disease and to develop therapies based on this understanding. The goal of this study was to develop cell-permeable (CP-) forms of the RUNT-related transcription factor 3, RUNX3-a candidate tumor suppressor implicated in gastric and other epithelial cancers-to study the therapeutic potential of RUNX3 in the treatment of gastric cancer. EXPERIMENTAL
DESIGN: We developed novel macromolecule transduction domains (MTD) which were tested for the ability to promote protein uptake by mammalian cells and tissues and used to deliver of biologically active RUNX3 into human gastric cancer cells. The therapeutic potential CP-RUNX3 was tested in the NCI-N87 human tumor xenograft animal model.
RESULTS: RUNX3 fusion proteins, HM(57)R and HM(85)R, containing hydrophobic MTDs enter gastric cancer cells and suppress cell phenotypes (e.g., cell-cycle progression, wounded monolayer healing, and survival) and induce changes in biomarker expression (e.g., p21(Waf1) and VEGF) consistent with previously described effects of RUNX3 on TGF-β signaling. CP-RUNX3 also suppressed the growth of subcutaneous human gastric tumor xenografts. The therapeutic response was comparable with studies augmenting RUNX3 gene expression in tumor cell lines; however, the protein was most active when administered locally, rather than systemically (i.e., intravenously).
CONCLUSIONS: These results provide further evidence that RUNX3 can function as a tumor suppressor and suggest that practical methods to augment RUNX3 function could be useful in treating of some types of gastric cancer.

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Year:  2012        PMID: 23230322      PMCID: PMC4039637          DOI: 10.1158/1078-0432.CCR-12-2692

Source DB:  PubMed          Journal:  Clin Cancer Res        ISSN: 1078-0432            Impact factor:   12.531


  35 in total

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Authors:  L S H Chuang; Y Ito
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Review 2.  Benefit of adjuvant chemotherapy for resectable gastric cancer: a meta-analysis.

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3.  Hyperactivation of Stat3 in gp130 mutant mice promotes gastric hyperproliferation and desensitizes TGF-beta signaling.

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Journal:  Nat Med       Date:  2005-07-24       Impact factor: 53.440

4.  RUNX3, a novel tumor suppressor, is frequently inactivated in gastric cancer by protein mislocalization.

Authors:  Kosei Ito; Qiang Liu; Manuel Salto-Tellez; Takashi Yano; Kotaro Tada; Hiroshi Ida; Canhua Huang; Nilesh Shah; Masafumi Inoue; Andrea Rajnakova; Kum Chew Hiong; Bee Keow Peh; Hwan Chour Han; Tomoko Ito; Ming Teh; Khay Guan Yeoh; Yoshiaki Ito
Journal:  Cancer Res       Date:  2005-09-01       Impact factor: 12.701

5.  RUNX3 suppresses gastric epithelial cell growth by inducing p21(WAF1/Cip1) expression in cooperation with transforming growth factor {beta}-activated SMAD.

Authors:  Xin-Zi Chi; Jeung-Ook Yang; Kwang-Youl Lee; Kosei Ito; Chohei Sakakura; Qing-Lin Li; Hye-Ryun Kim; Eun-Jeung Cha; Yong-Hee Lee; Atsushi Kaneda; Toshikazu Ushijima; Wun-Jae Kim; Yoshiaki Ito; Suk-Chul Bae
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Review 6.  Genomic and epigenetic profiles of gastric cancer: potential diagnostic and therapeutic applications.

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Journal:  Surg Today       Date:  2010-12-30       Impact factor: 2.549

7.  In vivo islet protection by a nuclear import inhibitor in a mouse model of type 1 diabetes.

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8.  Gastric cancer.

Authors:  Henk H Hartgrink; Edwin P M Jansen; Nicole C T van Grieken; Cornelis J H van de Velde
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Review 9.  Molecular pathology of RUNX3 in human carcinogenesis.

Authors:  Manish Mani Subramaniam; Jason Yongsheng Chan; Khay Guan Yeoh; Timothy Quek; Kosei Ito; Manuel Salto-Tellez
Journal:  Biochim Biophys Acta       Date:  2009-08-12

10.  Absence of Runx3 expression in normal gastrointestinal epithelium calls into question its tumour suppressor function.

Authors:  Ditsa Levanon; Yael Bernstein; Varda Negreanu; Karen Rae Bone; Amir Pozner; Raya Eilam; Joseph Lotem; Ori Brenner; Yoram Groner
Journal:  EMBO Mol Med       Date:  2011-08-08       Impact factor: 12.137

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  3 in total

1.  Partial somatic to stem cell transformations induced by cell-permeable reprogramming factors.

Authors:  Junghee Lim; Junghee Kim; Jinsun Kang; Daewoong Jo
Journal:  Sci Rep       Date:  2014-03-12       Impact factor: 4.379

2.  CpG-oligodeoxynucleotides suppress the proliferation of A549 lung adenocarcinoma cells via toll-like receptor 9 signaling and upregulation of Runt-related transcription factor 3 expression.

Authors:  Prince Amoah Barnie; Pan Zhang; Ping Lu; Xiaobo Chen; Zhaoliang Su; Shengjun Wang; Huaxi Xu
Journal:  Biomed Rep       Date:  2014-03-18

3.  Cell-permeable parkin proteins suppress Parkinson disease-associated phenotypes in cultured cells and animals.

Authors:  Tam Duong; Jaetaek Kim; H Earl Ruley; Daewoong Jo
Journal:  PLoS One       Date:  2014-07-14       Impact factor: 3.240

  3 in total

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