Literature DB >> 23192725

Pharmacokinetics of midazolam in resuscitated patients treated with moderate hypothermia.

Diane E T Bastiaans1, Eleonora L Swart, Jesse P van Akkeren, Luc J J Derijks.   

Abstract

BACKGROUND: Patients who remain comatose after resuscitation are treated with moderate hypothermia. Little is known about the pharmacokinetics of drugs in patients who are treated with moderate hypothermia.
OBJECTIVE: We investigated the pharmacokinetics of midazolam in resuscitated patients treated with moderate hypothermia in comparison to normothermic and non-resuscitated patients.
SETTING: This study was performed on the ICU of a Dutch non-academic hospital. The study population consisted of nine patients in the hypothermic group and eight patients in the control group.
METHOD: The resuscitated patients were cooled to a target temperature of 33 °C and rewarmed 24 h after start of cooling. Midazolam was given as continuous infusion. The control group consisted of non-resuscitated ICU-patients who were treated with midazolam as sedative. Plasma concentration-time data were collected for midazolam and its metabolites. MAIN OUTCOME MEASURE: Non-linear mixed effect modelling was used to analyze midazolam population pharmacokinetics and identify possible covariates.
RESULTS: A two-compartment pharmacokinetic model best describes the data. The pharmacokinetic models of the investigated groups are not significantly different. Pharmacokinetic parameter estimates for midazolam for the hypothermic group are a body clearance (CL) of 12.6 l/h, an apparent volume of the central compartment (V1) of 19.1 l, an apparent volume of the peripheral compartment (V2) of 108 l and an intercompartmental clearance (Q) of 18.4 l/h. Estimated parameters for the control group are CL of 14.2 l/h, a V1 of 15.7 l, a V2 of 171 l and Q of 25.6 l/h. In the covariate analysis, body temperature did not significantly improve the model.
CONCLUSION: We found no significant difference in the pharmacokinetics of midazolam between resuscitated patients treated with hypothermia during 24 h and the control group.

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Year:  2012        PMID: 23192725     DOI: 10.1007/s11096-012-9725-0

Source DB:  PubMed          Journal:  Int J Clin Pharm


  10 in total

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Authors: 
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5.  Treatment of comatose survivors of out-of-hospital cardiac arrest with induced hypothermia.

Authors:  Stephen A Bernard; Timothy W Gray; Michael D Buist; Bruce M Jones; William Silvester; Geoff Gutteridge; Karen Smith
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6.  Effect of atorvastatin and fluvastatin on the metabolism of midazolam by cytochrome P450 in vitro.

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7.  Prolonged sedation due to accumulation of conjugated metabolites of midazolam.

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8.  Comparative population pharmacokinetics of lorazepam and midazolam during long-term continuous infusion in critically ill patients.

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9.  Biphasic concentration change during continuous midazolam administration in brain-injured patients undergoing therapeutic moderate hypothermia.

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  10 in total
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1.  Hypoalbuminaemia and decreased midazolam clearance in terminally ill adult patients, an inflammatory effect?

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Review 2.  Effect of Hypothermia and Targeted Temperature Management on Drug Disposition and Response Following Cardiac Arrest: A Comprehensive Review of Preclinical and Clinical Investigations.

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Review 3.  A Physiology-Based Pharmacokinetic Framework to Support Drug Development and Dose Precision During Therapeutic Hypothermia in Neonates.

Authors:  Anne Smits; Pieter Annaert; Steven Van Cruchten; Karel Allegaert
Journal:  Front Pharmacol       Date:  2020-05-13       Impact factor: 5.810

  3 in total

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