Literature DB >> 23068967

An experimental model of contact dermatitis: evaluation of the oxidative profile of Wistar rats treated with free and nanoencapsulated clobetasol.

Jeandre Augusto dos Santos Jaques1, João Felipe Peres Rezer, Jader Betsch Ruchel, Viviane do Carmo Gonçalves Souza, Kelly de Vargas Pinheiro, Karine Bizzi Schlemmer, Josiane Bizzi Schlemmer, Tatiana Montagner Dalcin Bertoldo, Nara Maria Beck Martins, Cláudia de Mello Bertoncheli, Márcia Camponogara Fontana, Ruy Carlos Ruver Beck, Daniela Bitencourt Rosa Leal.   

Abstract

OBJECTIVE: An experimental animal model of contact dermatitis (CD) was used to investigate the effects of free and nanoencapsulated clobetasol propionate on the skin and on the oxidative profile of liver tissue.
METHODS: Female Wistar rats were divided into six groups, each containing eight rats. The first group, control (C), was sensitized with solid vaseline. Group 2, (CD), was sensitized with 5% NiSO(4). Groups 3 and 4 were sensitized with 5% NiSO(4) and treated with free (FC) and nanoencapsulated (NC) clobetasol (0.42 mg/g), respectively, daily for 5 days. Group 5 was treated with nanoencapsulated clobetasol (0.42 mg/g) on days 1, 3, and 5 (C135) and group 6 received a hydrogel containing empty nanoparticles (NP) daily for 5 days. Thiobarbituric acid reactive substances (TBARS), carbonyl levels, non-protein sulfhydryl groups (NPSH) and catalase activity were measured in liver homogenates.
RESULTS: A significant increase was observed in the levels of TBARS, NPSH, and catalase activity for the groups CD and NP. DISCUSSION: Our results suggest that both NiSO(4) sensitization and NP administration induced oxidation of cellular lipids and activated the antioxidant enzyme catalase to protect from this damage. These results also indicated that daily treatment with the free and nanoencapsulated clobetasol, as well as treatment with the nanoencapsulated clobetasol every other day, were able to prevent these redox alterations and protect against histological damage.

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Year:  2012        PMID: 23068967      PMCID: PMC6837319          DOI: 10.1179/1351000212Y.0000000024

Source DB:  PubMed          Journal:  Redox Rep        ISSN: 1351-0002            Impact factor:   4.412


  42 in total

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