Literature DB >> 22990063

Effects of prolyl 4-hydroxylase inhibitor on bladder function, bladder hypertrophy and collagen subtypes in a rat model with partial bladder outlet obstruction.

Jae Min Chung1, Min Jung Jung, Sang-Jin Lee, Sang Don Lee.   

Abstract

OBJECTIVE: To investigate the effects of prolyl 4-hydroxylase (P4H) inhibitor on the bladder in rats with partial bladder outlet obstruction.
METHODS: Forty-five female Sprague-Dawley rats were divided into 3 groups; partial bladder outlet obstruction with P4H inhibitor 20 mg/kg (groups A1, A2, and A4, each n = 5), partial bladder outlet obstruction with normal saline (groups B1, B2, and B4, each n = 5), and normal control (groups C1, C2, and C4, each n = 5). After partial bladder outlet obstruction for 1, 2, and 4 weeks in the groups A and B, respectively, the inhibitor or normal saline were administered orally at the indicated dosage once a day for 2 weeks. After either 3, 4, or 6 weeks, the bladders were removed after cystometry.
RESULTS: The pressure and volume parameters from the cystometry and the muscle thickness from the Masson trichrome staining of groups A and B increased significantly compared to those in group C (P < .05), and those in group A were significantly lower than those of group B (P < .05). Based on immunohistochemical staining and reverse transcription-polymerase chain reaction, P4H expression in groups A and B was increased compared with that in group C. Furthermore, P4H expression showed a larger decrease in group A compared to that in group B. Collagens I and III protein expressions increased with partial bladder outlet obstruction in comparison with that of group C, and expression in group A was marginally decreased compared with expression in group B.
CONCLUSION: Our data suggest that the P4H inhibitor may improve bladder function and reduce the bladder fibrosis caused by partial bladder outlet obstruction.
Copyright © 2012 Elsevier Inc. All rights reserved.

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Year:  2012        PMID: 22990063     DOI: 10.1016/j.urology.2012.07.019

Source DB:  PubMed          Journal:  Urology        ISSN: 0090-4295            Impact factor:   2.649


  4 in total

1.  Bladder fibrosis during outlet obstruction is triggered through the NLRP3 inflammasome and the production of IL-1β.

Authors:  Francis M Hughes; Stephanie J Sexton; Huixia Jin; Vihasa Govada; J Todd Purves
Journal:  Am J Physiol Renal Physiol       Date:  2017-06-07

2.  Inhibition of hypoxia-inducible factor-prolyl hydroxylation protects from cyclophosphamide-induced bladder injury and urinary dysfunction.

Authors:  Douglass B Clayton; Ching Man Carmen Tong; Belinda Li; Abby S Taylor; Shuvro De; Matthew D Mason; Anne G Dudley; Olena Davidoff; Hanako Kobayashi; Volker H Haase
Journal:  Am J Physiol Renal Physiol       Date:  2022-05-02

3.  Effect of Sulforaphane on Bladder Compliance in a Rat Model of Partial Bladder Outlet Obstruction.

Authors:  Chong Liu; Xiang Wan; Meng Gu; Yanbo Chen; Zhikang Cai; Juan Zhou; Qi Chen; Zhong Wang
Journal:  Oxid Med Cell Longev       Date:  2019-06-18       Impact factor: 6.543

4.  Combination phosphodiesterase type 4 inhibitor and phosphodiesterase type 5 inhibitor treatment reduces non-voiding contraction in a rat model of overactive bladder.

Authors:  Brian M Balog; Abhilasha Tangada; Pooja Sheth; Qi-Xiang Song; Bruna M Couri; Leah L Porras; Gary G Deng; Margot S Damaser
Journal:  PLoS One       Date:  2019-08-28       Impact factor: 3.240

  4 in total

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