Literature DB >> 22981433

A histopathologic basis for surgical debridement to promote healing of venous ulcers.

Sheila N Blumberg1, Jason Maggi, Jonathan Melamed, Michael Golinko, Frank Ross, Weiliam Chen.   

Abstract

BACKGROUND: Pathologic analysis of deep tissue obtained during debridement of venous ulcers is often unnoticed in its importance. We previously reported pathologic findings on 139 patients with venous ulcers. The objective of this study was to correlate the pathologic findings in venous ulcers with wound healing to establish a negative margin for debridement. STUDY
DESIGN: Consecutive patients with a lower extremity venous ulcer present for at least 4 weeks, presenting to a single wound healing center, were included. Wounds underwent aggressive surgical debridement beyond the subcutaneous level until judged to have a viable base. Specimens were scored based on cellularity, vascularity, collagen composition, inflammation, and dense fibrosis, with a highest possible score of 13. Healing was the primary outcome for analysis.
RESULTS: Of the 26 patients who met inclusion criteria, only 50% of them (13 patients) with a total of 18 venous ulcers underwent surgical debridement available for pathologic analysis. Mean ulcer area was 34.7 cm(2) at initial presentation, and 89% of patients had a continuous positive healing curve as measured by decreasing wound area (from 34.7 cm(2) to 14.3 cm(2)). However, specimens with dense fibrosis, decreased cellularity, mature collagen, and pathology score less than 10 were predominantly nonhealing ulcers.
CONCLUSIONS: Presence of dense fibrosis and high levels of mature collagen in deep tissue specimens are significant correlative factors in nonhealing of venous ulcers. We recommend deep debridement on all venous ulcers that are refractory to healing until the level of absence of dense fibrosis and mature collagen is reached to promote venous ulcer healing.
Copyright © 2012 American College of Surgeons. Published by Elsevier Inc. All rights reserved.

Entities:  

Mesh:

Year:  2012        PMID: 22981433     DOI: 10.1016/j.jamcollsurg.2012.08.008

Source DB:  PubMed          Journal:  J Am Coll Surg        ISSN: 1072-7515            Impact factor:   6.113


  5 in total

1.  Trichloroacetic acid (80%) as a chemical debridement method for chronic venous leg ulcers-A pilot study.

Authors:  Rita R Pinheiro; Bruno Duarte; Joana Cabete
Journal:  Int Wound J       Date:  2018-01-15       Impact factor: 3.315

2.  A bioengineered living cell construct activates metallothionein/zinc/MMP8 and inhibits TGFβ to stimulate remodeling of fibrotic venous leg ulcers.

Authors:  Rivka C Stone; Olivera Stojadinovic; Andrew P Sawaya; George D Glinos; Linsey E Lindley; Irena Pastar; Evangelos Badiavas; Marjana Tomic-Canic
Journal:  Wound Repair Regen       Date:  2019-12-04       Impact factor: 3.617

3.  Integrative small and long RNA omics analysis of human healing and nonhealing wounds discovers cooperating microRNAs as therapeutic targets.

Authors:  Zhuang Liu; Letian Zhang; Maria A Toma; Dongqing Li; Xiaowei Bian; Irena Pastar; Marjana Tomic-Canic; Pehr Sommar; Ning Xu Landén
Journal:  Elife       Date:  2022-08-12       Impact factor: 8.713

4.  Mesenchymal stromal cells prevent bleomycin-induced lung and skin fibrosis in aged mice and restore wound healing.

Authors:  Gustavo A Rubio; Sharon J Elliot; Tongyu C Wikramanayake; Xiaomei Xia; Simone Pereira-Simon; Seth R Thaller; George D Glinos; Ivan Jozic; Penelope Hirt; Irena Pastar; Marjana Tomic-Canic; Marilyn K Glassberg
Journal:  J Cell Physiol       Date:  2018-02-28       Impact factor: 6.384

Review 5.  Genomics of Human Fibrotic Diseases: Disordered Wound Healing Response.

Authors:  Rivka C Stone; Vivien Chen; Jamie Burgess; Sukhmani Pannu; Marjana Tomic-Canic
Journal:  Int J Mol Sci       Date:  2020-11-14       Impact factor: 5.923

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.