Literature DB >> 22972413

The effects of perinatal fluoxetine treatment on the circadian system of the adult mouse.

Veronika Kiryanova1, Victoria M Smith, Richard H Dyck, Michael C Antle.   

Abstract

RATIONAL: Depression is prevalent among women of childbearing age and is frequently treated with selective serotonin reuptake inhibitors (SSRIs). As some SSRIs, such as fluoxetine (Flx), can cross the placenta, it is possible that the neurodevelopment of the fetus may be affected, leading to altered behavior in adulthood.
OBJECTIVES: In this study, we examined the effects of perinatal Flx exposure on the subsequent expression of circadian rhythms in adult mice.
METHODS: Dams were treated with 25 mg/kg/day Flx in their drinking water from embryonic day 15 to postnatal day 12. Circadian organization of wheel running rhythms and phase shifts to photic and non-photic stimuli were assessed in the offspring starting at 6 weeks of age.
RESULTS: We found that perinatal Flx exposure led to larger light-induced phase advances (1.19 ± 0.51 vs. 0.55 ± 0.25 h), smaller phase advances to the serotonin agonist 8-OH-DPAT during the mid-subjective day (0.44 ± 0.15 vs. 0.70 ± 0.17 h), and a shorter free-running period in constant darkness (23.47 ± 0.13 vs. 23.64 ± 0.13 h).
CONCLUSIONS: These results suggest that perinatal exposure to SSRIs may have consequences for the functioning of the circadian system later in life.

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Year:  2012        PMID: 22972413     DOI: 10.1007/s00213-012-2861-3

Source DB:  PubMed          Journal:  Psychopharmacology (Berl)        ISSN: 0033-3158            Impact factor:   4.530


  54 in total

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Journal:  Neuropharmacology       Date:  2011-09-24       Impact factor: 5.250

2.  Increasing use of antidepressants in pregnancy.

Authors:  William O Cooper; Mary E Willy; Stephen J Pont; Wayne A Ray
Journal:  Am J Obstet Gynecol       Date:  2007-06       Impact factor: 8.661

Review 3.  Serotonin as a developmental signal.

Authors:  P M Whitaker-Azmitia; M Druse; P Walker; J M Lauder
Journal:  Behav Brain Res       Date:  1996       Impact factor: 3.332

4.  Serotonergic potentiation of photic phase shifts of the circadian activity rhythm.

Authors:  M A Rea; J Barrera; J D Glass; R L Gannon
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5.  Early intervention with fluoxetine reverses abnormalities in the serotonergic system and behavior of rats exposed prenatally to dexamethasone.

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6.  Neonatal exposure to fluoxetine and fluvoxamine alteres spine density in mouse hippocampal CA1 pyramidal neurons.

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10.  Depletion of brain serotonin by 5,7-DHT modifies hamster circadian rhythm response to light.

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2.  Circadian behavior of adult mice exposed to stress and fluoxetine during development.

Authors:  Veronika Kiryanova; Victoria M Smith; Richard H Dyck; Michael C Antle
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3.  Transient postnatal fluoxetine decreases brain concentrations of 20-HETE and 15-epi-LXA4, arachidonic acid metabolites in adult mice.

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4.  Hippocampal HDAC4 contributes to postnatal fluoxetine-evoked depression-like behavior.

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5.  Does Infant Reactivity Moderate the Association Between Antenatal Maternal Depression and Infant Sleep?

Authors:  Elena Netsi; Marinus H van IJzendoorn; Marian J Bakermans-Kranenburg; Katharina Wulff; Pauline W Jansen; Vincent W V Jaddoe; Frank C Verhulst; Henning Tiemeier; Paul G Ramchandani
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6.  Perinatal fluoxetine exposure disrupts the circadian response to a phase-shifting challenge in female rats.

Authors:  Danielle J Houwing; Jolien de Waard; Anouschka S Ramsteijn; Tom Woelders; Sietse F de Boer; Emma J Wams; Jocelien D A Olivier
Journal:  Psychopharmacology (Berl)       Date:  2020-06-12       Impact factor: 4.530

  6 in total

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