Literature DB >> 22968213

Therapeutic effects of neuregulin-1 gene transduction in rats with myocardial infarction.

Jian Xiao1, Bingong Li, Zeqi Zheng, Menghong Wang, Jingtian Peng, Yong Li, Zhe Li.   

Abstract

AIM: In this study, we investigated whether lentivirus-mediated gene transduction improves the cardiac function in rats with myocardial infarction and the mechanisms involved. METHODS AND
RESULTS: Briefly, lentivirus carrying human neuregulin-1 (hNRG-1) gene was injected into infarcted myocardium of rats. Four weeks later, lentivirus-mediated gene transduction promoted hNRG-1 gene and protein expression. Overexpression of hNRG-1 increased the number of microvessels in the ischemic myocardium and decreased the number of terminal deoxynucleotidyl transferase-mediated dUTP nick-end labeling (TUNEL)-stained positive cells. Furthermore, qRT-PCR assay showed that hNRG-1 was capable of increasing the expression of bcl-2 and VEGF-A while decreasing the expression of bax. Western blot analysis suggested that overexpression of hNRG-1 activated the PI3K/Akt pathway and increased the phosphorylation of Akt and eNOS.
CONCLUSION: These findings suggest that NRG-1 gene transduction can improve cardiac function by promoting angiogenesis and preventing apoptosis.
© 2012 Wolters Kluwer Health | Lippincott Williams & Wilkins.

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Year:  2012        PMID: 22968213     DOI: 10.1097/MCA.0b013e32835877da

Source DB:  PubMed          Journal:  Coron Artery Dis        ISSN: 0954-6928            Impact factor:   1.439


  14 in total

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9.  Neuregulin‑1 protects cardiac function in septic rats through multiple targets based on endothelial cells.

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