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Literature DB >> 22942220

Counterpoint: alterations in airway smooth muscle phenotype do not cause airway hyperresponsiveness in asthma.

Peter D Paré¹, Wayne Mitzner.

Abstract

Entities: Disease

Mesh：

Year: 2012 PMID： 22942220 PMCID： PMC4596735 DOI： 10.1152/japplphysiol.00483.2012a

Source DB: PubMed Journal: J Appl Physiol (1985) ISSN： 0161-7567

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32 in total

1. Airway structural alterations selectively associated with severe asthma.

Authors: Laurent Benayoun; Anne Druilhe; Marie-Christine Dombret; Michel Aubier; Marina Pretolani
Journal: Am J Respir Crit Care Med Date: 2003-01-16 Impact factor: 21.405

2. Extracellular matrix proteins modulate asthmatic airway smooth muscle cell proliferation via an autocrine mechanism.

Authors: Peter R A Johnson; Janette K Burgess; P Anne Underwood; Wendy Au; Maree H Poniris; Michael Tamm; Qi Ge; Michael Roth; Judith L Black
Journal: J Allergy Clin Immunol Date: 2004-04 Impact factor: 10.793

3. A 'Good' muscle in a 'Bad' environment: the importance of airway smooth muscle force adaptation to airway hyperresponsiveness.

Authors: Ynuk Bossé; David G Chapman; Peter D Paré; Gregory G King; Cheryl M Salome
Journal: Respir Physiol Neurobiol Date: 2011-09-14 Impact factor: 1.931

1. Last Word on Counterpoint: Alterations in airway smooth muscle phenotype do not cause airway hyperresponsiveness in asthma.

Authors: Peter D Paré; Wayne Mitzner
Journal: J Appl Physiol (1985) Date: 2012-09-01

Review 2. Systems physiology of the airways in health and obstructive pulmonary disease.

Authors: Jason H T Bates
Journal: Wiley Interdiscip Rev Syst Biol Med Date: 2016-06-24

2 in total

Counterpoint: alterations in airway smooth muscle phenotype do not cause airway hyperresponsiveness in asthma.

1. Airway structural alterations selectively associated with severe asthma.

2. Extracellular matrix proteins modulate asthmatic airway smooth muscle cell proliferation via an autocrine mechanism.

3. A 'Good' muscle in a 'Bad' environment: the importance of airway smooth muscle force adaptation to airway hyperresponsiveness.

4. Abnormalities in airway smooth muscle in fatal asthma.

5. Hypothesis: excessive bronchoconstriction in asthma is due to decreased airway elastance.

6. Changes in biophysical and biochemical properties of single bronchial smooth muscle cells from asthmatic subjects.

7. Expression of connective tissue growth factor in asthmatic airway smooth muscle cells.

8. Abnormalities in airway smooth muscle in fatal asthma. A comparison between trachea and bronchus.

9. Dysfunctional interaction of C/EBPalpha and the glucocorticoid receptor in asthmatic bronchial smooth-muscle cells.

10. The pivotal role of airway smooth muscle in asthma pathophysiology.

1. Last Word on Counterpoint: Alterations in airway smooth muscle phenotype do not cause airway hyperresponsiveness in asthma.

Review 2. Systems physiology of the airways in health and obstructive pulmonary disease.