Literature DB >> 22921817

A combination of increased Rho kinase activity and N-terminal pro-B-type natriuretic peptide predicts worse cardiovascular outcome in patients with acute coronary syndrome.

Ming Dong1, James K Liao, Bryan Yan, Ruijie Li, Mang Zhang, Cheuk-Man Yu.   

Abstract

BACKGROUND: Recent experimental evidence suggests that the Rho/Rho-kinase (ROCK) system may play an important role in the pathogenesis of acute coronary syndrome (ACS) but there are little clinical data. This study examined if ROCK activity is increased in patients with acute coronary syndrome and if ROCK activity predicts long-term cardiovascular event.
METHOD: Blood samples were collected from 188 patients within 12h after admission for ACS (53% men; aged 70 ± 13) and from 61 control subject. The main outcome measures were all cause mortality, readmission with ACS or congestive heart failure (CHF) from presentation within around 2 years (mean:14.4 ± 7.2 months; range: 0.5 to 26 months).
RESULTS: ROCK activity increased in ST elevation myocardial infarction (STEMI, n=90) (3.33 ± 0.93), non-STEMI (NSTEMI, n=68) (3.37 ± 1.04) and unstable angina (UA, n=30) (2.53 ± 0.59) groups when compared with disease controls (n=31) (2.06 ± 0.38, all p<0.001) and healthy controls (n=30) (1.54 ± 0.43, all p<0.001). There were 24 deaths, 34 readmissions with ACS and 15 admissions with CHF within 2 years. Patients with a high N-terminal pro-B-type natriuretic peptide (NT-proBNP) and high ROCK activity on admission had a five-fold risk of a cardiovascular event (RR: 5.156; 95% CI: 2.180-12.191) when compared to those with low NT-proBNP and low ROCK activity.
CONCLUSION: ROCK activity was increased in patients with ACS, particularly in those with myocardial infarction. The combined usage of both ROCK activity and NT-proBNP might identify a subset of ACS patients at particularly high risk.
Copyright © 2012 Elsevier Ireland Ltd. All rights reserved.

Entities:  

Keywords:  ACS; ANOVA; Acute coronary syndrome; BH; BMI; BW; Body height; Body mass index; Body weight; C-reactive protein; CHD; CHF; CIHD; CPK; CRP; CVA; Cardiac troponin T; Cardio vascular accidents; Congestive heart failure; Coronary heart disease; Coronary ischemic heart disease; Creatine phosphokinase; DBP; DM; Diabetes mellitus; Diastolic blood pressure; Estimated Glomerular Filtration Rate; GRACE; Global Registry of Acute Coronary Events; HDL-C; HR; Heart rate; High-density lipoprotein cholesterol; LDL-C; LVEF; Left ventricular ejection fraction; Low-density lipoprotein cholesterol; N-terminal pro-B-type natriuretic peptide; NSTEMI; NT-proBNP; Non-ST elevation myocardial infarction; One-way analysis of variance; PCI; Percutaneous coronary intervention; ROCK; ROCK activity; Rho kinase; Rho kinase activity; SBP; ST elevation myocardial infarction; STEMI; Systolic blood pressure; TC; TG; Total cholesterol; Triglycerides; UA; Unstable angina; WBC; White blood cells; cTnT; eGFR

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Year:  2012        PMID: 22921817      PMCID: PMC3807084          DOI: 10.1016/j.ijcard.2012.07.007

Source DB:  PubMed          Journal:  Int J Cardiol        ISSN: 0167-5273            Impact factor:   4.164


  40 in total

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9.  Determinants and prognostic impact of heart failure complicating acute coronary syndromes: observations from the Global Registry of Acute Coronary Events (GRACE).

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10.  Rho-associated protein kinase contributes to early atherosclerotic lesion formation in mice.

Authors:  Ziad Mallat; Andrea Gojova; Vincent Sauzeau; Valérie Brun; Jean-Sébastien Silvestre; Bruno Esposito; Régine Merval; Hervé Groux; Gervaise Loirand; Alain Tedgui
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Journal:  In Vivo       Date:  2018 Mar-Apr       Impact factor: 2.155

2.  Elevated plasma interleukin-37 playing an important role in acute coronary syndrome through suppression of ROCK activation.

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