Literature DB >> 22914838

Peritrigonal and temporo-occipital heterotopia with corpus callosum and cerebellar dysgenesis.

Tiziana Pisano1, A James Barkovich, Richard J Leventer, Waney Squier, Ingrid E Scheffer, Elena Parrini, Susan Blaser, Carla Marini, Stephen Robertson, Gaetano Tortorella, Felix Rosenow, Pierre Thomas, George McGillivray, Eva Andermann, Frederick Andermann, Samuel F Berkovic, William B Dobyns, Renzo Guerrini.   

Abstract

OBJECTIVE: To describe a homogeneous subtype of periventricular nodular heterotopia (PNH) as part of a newly defined malformation complex.
METHODS: Observational study including review of brain MRI and clinical findings of a cohort of 50 patients with PNH in the temporo-occipital horns and trigones, mutation analysis of the FLNA gene, and anatomopathologic study of a fetal brain.
RESULTS: There were 28 females and 22 males. All were sporadic with the exception of an affected mother and son. Epilepsy occurred in 62%, cerebellar signs in 56%, cognitive impairment in 56%, and autism in 12%. Seventy percent were referred within the 3rd year of life. Imaging revealed a normal cerebral cortex in 76% and abnormal cortical folding in 24%. In all patients the hippocampi were under-rotated and in 10% they merged with the heterotopia. Cerebellar dysgenesis was observed in 84% and a hypoplastic corpus callosum in 60%. There was no gender bias or uneven gender distribution of clinical and anatomic severity. No mutations of FLNA occurred in 33 individuals examined. Heterotopia in the fetal brain revealed cytoarchitectonic characteristics similar to those associated with FLNA mutations; cortical pathology was not typical of polymicrogyria. Cerebellar involvement was more severe and the hippocampi appeared simple and under-rotated.
CONCLUSIONS: This series delineates a malformation complex in which PNH in the trigones and occipito-temporal horns is associated with hippocampal, corpus callosum, and cerebellar dysgenesis. This subtype of PNH is distinct from classic PNH caused by FLNA mutations.

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Year:  2012        PMID: 22914838      PMCID: PMC3440449          DOI: 10.1212/WNL.0b013e31826aac88

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  22 in total

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4.  Bilateral periventricular nodular heterotopia due to filamin 1 gene mutation: widespread glomeruloid microvascular anomaly and dysplastic cytoarchitecture in the cerebral cortex.

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8.  Mutations in the X-linked filamin 1 gene cause periventricular nodular heterotopia in males as well as in females.

Authors:  V L Sheen; P H Dixon; J W Fox; S E Hong; L Kinton; S M Sisodiya; J S Duncan; F Dubeau; I E Scheffer; S C Schachter; A Wilner; R Henchy; P Crino; K Kamuro; F DiMario; M Berg; R Kuzniecky; A J Cole; E Bromfield; M Biber; D Schomer; J Wheless; K Silver; G H Mochida; S F Berkovic; F Andermann; E Andermann; W B Dobyns; N W Wood; C A Walsh
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9.  Periventricular nodular heterotopia with overlying polymicrogyria.

Authors:  Gretchen Wieck; Richard J Leventer; Waney M Squier; An Jansen; Eva Andermann; Francois Dubeau; Anna Ramazzotti; Renzo Guerrini; William B Dobyns
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10.  The autism susceptibility gene met regulates zebrafish cerebellar development and facial motor neuron migration.

Authors:  Gina E Elsen; Louis Y Choi; Victoria E Prince; Robert K Ho
Journal:  Dev Biol       Date:  2009-09-02       Impact factor: 3.582

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5.  Quantitative assessment of corpus callosum morphology in periventricular nodular heterotopia.

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Review 6.  Genetic Basis of Brain Malformations.

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Journal:  Mol Syndromol       Date:  2016-08-27

7.  Phenotypic and imaging features of FLNA-negative patients with bilateral periventricular nodular heterotopia and epilepsy.

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8.  De novo and inherited private variants in MAP1B in periventricular nodular heterotopia.

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9.  Integrity of the corpus callosum in patients with periventricular nodular heterotopia related epilepsy by FLNA mutation.

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10.  Midbrain-hindbrain malformations in patients with malformations of cortical development and epilepsy: a series of 220 patients.

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